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Early virus-host interactions dictate the course of a persistent infection

Early virus-host interactions dictate the course of a persistent infection

http://www.wikidata.org/entity/Q34876341

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Blockade of interferon Beta, but not interferon alpha, signaling controls persistent viral infection Current concepts on the pathogenesis of type 1 diabetes--considerations for attempts to prevent and reverse the disease.PTPN22 contributes to exhaustion of T lymphocytes during chronic viral infection.Alpha and Beta Type 1 Interferon Signaling: Passage for Diverse Biologic Outcomes.Residues K465 and G467 within the Cytoplasmic Domain of GP2 Play a Critical Role in the Persistence of Lymphocytic Choriomeningitis Virus in Mice.Evolutionary analysis of Old World arenaviruses reveals a major adaptive contribution of the viral polymerase.Efficacy of a Virus-Like Nanoparticle As Treatment for a Chronic Viral Infection Is Hindered by IRAK1 Regulation and Antibody Interference.Ancient Evolution of Mammarenaviruses: Adaptation via Changes in the L Protein and No Evidence for Host-Virus Codivergence.

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Early virus-host interactions dictate the course of a persistent infection

http://www.wikidata.org/entity/Q34876341

description

2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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type

label

Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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Early virus-host interactions dictate the course of a persistent infection

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JOURNAL.PPAT.1004588

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Early virus-host interactions dictate the course of a persistent infection

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Brian M Sullivan

http://www.w3.org/2001/XMLSchema#string

John R Teijaro

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Juan Carlos de la Torre

http://www.w3.org/2001/XMLSchema#string

Michael B A Oldstone

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P2860

Z proteins of New World arenaviruses bind RIG-I and interfere with type I interferon induction

Species-specific recognition of single-stranded RNA via toll-like receptor 7 and 8

NP and L proteins of lymphocytic choriomeningitis virus (LCMV) are sufficient for efficient transcription and replication of LCMV genomic RNA analogs

The complete nucleotide sequence of the gene coding for the human aldolase C

Viral immune evasion due to persistence of activated T cells without effector function

Cytosolic sensing of viruses

Immune responses to HCV and other hepatitis viruses

Slc15a4, a gene required for pDC sensing of TLR ligands, is required to control persistent viral infection

Interleukin-10 determines viral clearance or persistence in vivo

Site-specific antibodies define a cleavage site conserved among arenavirus GP-C glycoproteins

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e1004588

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scholarly article

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10.1371/JOURNAL.PPAT.1004588

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1

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11

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