Sequestration of aggregated low-density lipoproteins by macrophages.
about
Nuclear Molecular Imaging for Vulnerable Atherosclerotic PlaquesThe effect of particle agglomeration on the formation of a surface-connected compartment induced by hydroxyapatite nanoparticles in human monocyte-derived macrophagesMacrophages create an acidic extracellular hydrolytic compartment to digest aggregated lipoproteins.Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic miceLysosomal acid lipase: at the crossroads of normal and atherogenic cholesterol metabolismMacrophage cholesterol homeostasis and metabolic diseases: critical role of cholesteryl ester mobilization.LDL Receptor-Related Protein-1 (LRP1) Regulates Cholesterol Accumulation in Macrophages.In macrophages, HIV-1 assembles into an intracellular plasma membrane domain containing the tetraspanins CD81, CD9, and CD53.Size-selective uptake of colloidal low density lipoprotein aggregates by cultured white blood cellsCytomegalovirus in Plasma of Acute Coronary Syndrome Patients.Immune effector mechanisms implicated in atherosclerosis: from mice to humans.Loss of SR-A and CD36 activity reduces atherosclerotic lesion complexity without abrogating foam cell formation in hyperlipidemic mice.HIV-1 assembly in macrophagesCholesterol, cytokines and diseases.Contribution of monocyte-derived macrophages and smooth muscle cells to arterial foam cell formation.Regulation of gene expression in atherosclerosis: insights from microarray studies in monocytes/macrophages.An integrated approach for the mechanisms responsible for atherosclerotic plaque regressionEating the Dead to Keep Atherosclerosis at Bay.Internalization of SiO₂ nanoparticles by alveolar macrophages and lung epithelial cells and its modulation by the lung surfactant substitute Curosurf.Emerging roles of calpain proteolytic systems in macrophage cholesterol handling.Mechanism of cellular uptake of genotoxic silica nanoparticlesInflammation and atherosclerosis: disease modulating therapies.Lysosomes, cholesterol and atherosclerosis.Ceramide activation of RhoA/Rho kinase impairs actin polymerization during aggregated LDL catabolism.The cytoplasmic domain of the low density lipoprotein (LDL) receptor-related protein, but not that of the LDL receptor, triggers phagocytosis.Macrophage-specific expression of group IIA sPLA2 results in accelerated atherogenesis by increasing oxidative stress.Aggregated LDL and lipid dispersions induce lysosomal cholesteryl ester accumulation in macrophage foam cells.Formation of ceramide-enriched domains in lipid particles enhances the binding of apolipoprotein E
P2860
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P2860
Sequestration of aggregated low-density lipoproteins by macrophages.
description
2002 nî lūn-bûn
@nan
2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Sequestration of aggregated low-density lipoproteins by macrophages.
@ast
Sequestration of aggregated low-density lipoproteins by macrophages.
@en
Sequestration of aggregated low-density lipoproteins by macrophages.
@nl
type
label
Sequestration of aggregated low-density lipoproteins by macrophages.
@ast
Sequestration of aggregated low-density lipoproteins by macrophages.
@en
Sequestration of aggregated low-density lipoproteins by macrophages.
@nl
prefLabel
Sequestration of aggregated low-density lipoproteins by macrophages.
@ast
Sequestration of aggregated low-density lipoproteins by macrophages.
@en
Sequestration of aggregated low-density lipoproteins by macrophages.
@nl
P1476
Sequestration of aggregated low-density lipoproteins by macrophages.
@en
P2093
Howard S Kruth
P304
P356
10.1097/00041433-200210000-00003
P577
2002-10-01T00:00:00Z