Previous ethanol experience enhances synaptic plasticity of NMDA receptors in the ventral tegmental area.
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Overlapping patterns of brain activation to food and cocaine cues in cocaine abusers: association to striatal D2/D3 receptorsAlcohol-preferring P rats emit spontaneous 22-28 kHz ultrasonic vocalizations that are altered by acute and chronic alcohol experience.Long-term consequences of developmental alcohol exposure on brain structure and function: therapeutic benefits of physical activityNADPH Oxidase Isoform 2 (NOX2) Is Involved in Drug Addiction Vulnerability in Progeny Developmentally Exposed to Ethanol.Dynamic regulation of midbrain dopamine neuron activity: intrinsic, synaptic, and plasticity mechanismsPotential substrates for nicotine and alcohol interactions: a focus on the mesocorticolimbic dopamine system.Alcohol consumption induces global gene expression changes in VTA dopaminergic neuronsIs alcoholism learned? Insights from the fruit fly.Glutamatergic plasticity and alcohol dependence-induced alterations in reward, affect and cognition.New insights on neurobiological mechanisms underlying alcohol addiction.Effects of alcohol on the membrane excitability and synaptic transmission of medium spiny neurons in the nucleus accumbens.Effects of exposure to moderate levels of ethanol during prenatal brain development on dendritic length, branching, and spine density in the nucleus accumbens and dorsal striatum of adult ratsAcute and chronic effects of ethanol on learning-related synaptic plasticity.Glutamate plasticity woven through the progression to alcohol use disorder: a multi-circuit perspective.Synaptic plasticity of NMDA receptors: mechanisms and functional implications.Metaplasticity in human cortex.Associative Learning Drives the Formation of Silent Synapses in Neuronal Ensembles of the Nucleus Accumbens.Do specific NMDA receptor subunits act as gateways for addictive behaviors?Alcohol-dependent molecular adaptations of the NMDA receptor system.Repeated social defeat stress enhances glutamatergic synaptic plasticity in the VTA and cocaine place conditioning.L-type Ca²⁺ channel blockade with antihypertensive medication disrupts VTA synaptic plasticity and drug-associated contextual memory.Social deprivation enhances VTA synaptic plasticity and drug-induced contextual learning.Sensitization of ethanol-induced place preference as a result of up-regulation of type 1 inositol 1,4,5-trisphosphate receptors in mouse nucleus accumbens.Cooperative CRF and α1 Adrenergic Signaling in the VTA Promotes NMDA Plasticity and Drives Social Stress Enhancement of Cocaine Conditioning.
P2860
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P2860
Previous ethanol experience enhances synaptic plasticity of NMDA receptors in the ventral tegmental area.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Previous ethanol experience en ...... in the ventral tegmental area.
@ast
Previous ethanol experience en ...... in the ventral tegmental area.
@en
Previous ethanol experience en ...... in the ventral tegmental area.
@nl
type
label
Previous ethanol experience en ...... in the ventral tegmental area.
@ast
Previous ethanol experience en ...... in the ventral tegmental area.
@en
Previous ethanol experience en ...... in the ventral tegmental area.
@nl
prefLabel
Previous ethanol experience en ...... in the ventral tegmental area.
@ast
Previous ethanol experience en ...... in the ventral tegmental area.
@en
Previous ethanol experience en ...... in the ventral tegmental area.
@nl
P2093
P2860
P1476
Previous ethanol experience en ...... in the ventral tegmental area.
@en
P2093
Brian E Bernier
Hitoshi Morikawa
Leslie R Whitaker
P2860
P304
P356
10.1523/JNEUROSCI.5282-10.2011
P407
P577
2011-04-01T00:00:00Z