The Q175 mouse model of Huntington's disease shows gene dosage- and age-related decline in circadian rhythms of activity and sleep.
about
'The clocks that time us'--circadian rhythms in neurodegenerative disordersRole of vasoactive intestinal peptide in the light input to the circadian system.Sex Differences in Circadian Dysfunction in the BACHD Mouse Model of Huntington's DiseaseCOMPASS: Continuous Open Mouse Phenotyping of Activity and Sleep Status.Retinal Ganglion Cells and Circadian Rhythms in Alzheimer's Disease, Parkinson's Disease, and BeyondAttenuated pupillary light responses and downregulation of opsin expression parallel decline in circadian disruption in two different mouse models of Huntington's diseaseNeurodegeneration and the Circadian Clock.Neurocardiovascular deficits in the Q175 mouse model of Huntington's disease.Altered excitatory and inhibitory inputs to striatal medium-sized spiny neurons and cortical pyramidal neurons in the Q175 mouse model of Huntington's disease.Quantitative Electroencephalographic Analysis Provides an Early-Stage Indicator of Disease Onset and Progression in the zQ175 Knock-In Mouse Model of Huntington's Disease.Misaligned feeding impairs memories.Early-onset sleep defects in Drosophila models of Huntington's disease reflect alterations of PKA/CREB signaling.Sleep deficits but no metabolic deficits in premanifest Huntington's diseaseCircadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid.Hypothalamic alterations in Huntington's disease patients: comparison with genetic rodent models.Therapeutic strategies for circadian rhythm and sleep disturbances in Huntington disease.Chronic Paroxetine Treatment Prevents the Emergence of Abnormal Electroencephalogram Oscillations in Huntington's Disease Mice.An Automated Home-Cage System to Assess Learning and Performance of a Skilled Motor Task in a Mouse Model of Huntington's DiseasePossible use of a H3R antagonist for the management of nonmotor symptoms in the Q175 mouse model of Huntington's disease.Progressive axonal transport and synaptic protein changes correlate with behavioral and neuropathological abnormalities in the heterozygous Q175 KI mouse model of Huntington's disease.Plasma melatonin is reduced in Huntington's disease.Time-Restricted Feeding Improves Circadian Dysfunction as well as Motor Symptoms in the Q175 Mouse Model of Huntington’s Disease.Neurobiological Functions of the Period Circadian Clock 2 Gene, Per2.A Huntingtin Knockin Pig Model Recapitulates Features of Selective Neurodegeneration in Huntington's Disease.
P2860
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P2860
The Q175 mouse model of Huntington's disease shows gene dosage- and age-related decline in circadian rhythms of activity and sleep.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@ast
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@en
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@nl
type
label
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@ast
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@en
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@nl
prefLabel
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@ast
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@en
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@nl
P2860
P50
P1433
P1476
The Q175 mouse model of Huntin ...... rhythms of activity and sleep.
@en
P2093
Yingfei Wu
P2860
P304
P356
10.1371/JOURNAL.PONE.0069993
P407
P577
2013-07-30T00:00:00Z