Vibrio cholerae triggers SOS and mutagenesis in response to a wide range of antibiotics: a route towards multiresistance.
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Bacterial Responses and Genome Instability Induced by Subinhibitory Concentrations of AntibioticsA role for the bacterial GATC methylome in antibiotic stress survivalAntimicrobial-induced DNA damage and genomic instability in microbial pathogensAntibiotics trigger initiation of SCCmec transfer by inducing SOS responses.Integrons: past, present, and future.Antimicrobial Activity and Resistance: Influencing Factors.Genomic interplay in bacterial communities: implications for growth promoting practices in animal husbandry.Biological cost of pyocin production during the SOS response in Pseudomonas aeruginosa.Evidence for induction of integron-based antibiotic resistance by the SOS response in a clinical setting.Global transcriptome and physiological responses of Acinetobacter oleivorans DR1 exposed to distinct classes of antibiotics.Starvation, together with the SOS response, mediates high biofilm-specific tolerance to the fluoroquinolone ofloxacin.RpoS plays a central role in the SOS induction by sub-lethal aminoglycoside concentrations in Vibrio cholerae.Antimicrobial use in aquaculture re-examined: its relevance to antimicrobial resistance and to animal and human health.Diversity of Class 1 Integron Gene Cassette Rearrangements Selected under Antibiotic Pressure.Connecting environment and genome plasticity in the characterization of transformation-induced SOS regulation and carbon catabolite control of the Vibrio cholerae integron integrase.Antibiotic resistance shaping multi-level population biology of bacteria.Multiple Pathways of Genome Plasticity Leading to Development of Antibiotic Resistance.β-Lactam antibiotics promote bacterial mutagenesis via an RpoS-mediated reduction in replication fidelity.Antimicrobial resistance and virulence: a successful or deleterious association in the bacterial world?Characterization of the phd-doc and ccd toxin-antitoxin cassettes from Vibrio superintegrons.Systematically Altering Bacterial SOS Activity under Stress Reveals Therapeutic Strategies for Potentiating AntibioticsThe Stringent Response Promotes Antibiotic Resistance Dissemination by Regulating Integron Integrase Expression in Biofilms.DNA damage responses in prokaryotes: regulating gene expression, modulating growth patterns, and manipulating replication forks.Riboswitch control of induction of aminoglycoside resistance acetyl and adenyl-transferasesSpecificity in suppression of SOS expression by recA4162 and uvrD303.Identification of genes involved in low aminoglycoside-induced SOS response in Vibrio cholerae: a role for transcription stalling and Mfd helicase.A temporal proteome dynamics study reveals the molecular basis of induced phenotypic resistance in Mycobacterium smegmatis at sub-lethal rifampicin concentrationsBacterial stress responses as determinants of antimicrobial resistance.Mutation rate and the emergence of drug resistance in Mycobacterium tuberculosis.DNA damage repair and bacterial pathogensSelection of antibiotic resistance at very low antibiotic concentrations.Microbiological effects of sublethal levels of antibiotics.SOS, the formidable strategy of bacteria against aggressions.Effect of LexA on Chromosomal Integration of CTXϕ in Vibrio cholerae.Roles of Regulatory RNAs for Antibiotic Resistance in Bacteria and Their Potential Value as Novel Drug Targets.DNA-damaging agents induce the RecA-independent homologous recombination functions of integrating conjugative elements of the SXT/R391 family.Intracellular d-Serine Accumulation Promotes Genetic Diversity via Modulated Induction of RecA in Enterohemorrhagic Escherichia coli.Vibrio anguillarum Is Genetically and Phenotypically Unaffected by Long-Term Continuous Exposure to the Antibacterial Compound Tropodithietic Acid.Differential roles of antimicrobials in the acquisition of drug resistance through activation of the SOS response in Acinetobacter baumannii.Ciprofloxacin-Mediated Mutagenesis Is Suppressed by Subinhibitory Concentrations of Amikacin in Pseudomonas aeruginosa.
P2860
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P2860
Vibrio cholerae triggers SOS and mutagenesis in response to a wide range of antibiotics: a route towards multiresistance.
description
2011 nî lūn-bûn
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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@ast
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@en
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@nl
type
label
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@ast
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@en
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@nl
prefLabel
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@ast
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@en
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@nl
P2860
P356
P1476
Vibrio cholerae triggers SOS a ...... route towards multiresistance.
@en
P2093
Didier Mazel
Zeynep Baharoglu
P2860
P304
P356
10.1128/AAC.01549-10
P407
P577
2011-02-07T00:00:00Z