Selenite cataracts: activation of endoplasmic reticulum stress and loss of Nrf2/Keap1-dependent stress protection - Wikidata - wikimedia - TriplyDB

Selenite cataracts: activation of endoplasmic reticulum stress and loss of Nrf2/Keap1-dependent stress protection

Selenite cataracts: activation of endoplasmic reticulum stress and loss of Nrf2/Keap1-dependent stress protection

http://www.wikidata.org/entity/Q34940558

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Epigenetic regulation of Keap1-Nrf2 signaling The burden of trisomy 21 disrupts the proteostasis network in Down syndrome.Tangluoning, a traditional Chinese medicine, attenuates in vivo and in vitro diabetic peripheral neuropathy through modulation of PERK/Nrf2 pathway Ocular delivery of cyanidin-3-glycoside in liposomes and its prevention of selenite-induced oxidative stress.CpG site methylation in CRYAA promoter affect transcription factor Sp1 binding in human lens epithelial cells.Human βA3/A1-crystallin splicing mutation causes cataracts by activating the unfolded protein response and inducing apoptosis in differentiating lens fiber cells SLC4A11 depletion impairs NRF2 mediated antioxidant signaling and increases reactive oxygen species in human corneal endothelial cells during oxidative stress Mutations and mechanisms in congenital and age-related cataracts.The Role of DNA Methylation in Lens Development and Cataract Formation.Parkin elimination of mitochondria is important for maintenance of lens epithelial cell ROS levels and survival upon oxidative stress exposure.Nrf2 as a target for prevention of age-related and diabetic cataracts by against oxidative stress.Age-related cataracts: Role of unfolded protein response, Ca2+ mobilization, epigenetic DNA modifications, and loss of Nrf2/Keap1 dependent cytoprotection.Effect of senescence marker protein 30 on the proliferation and apoptosis of human lens epithelial cells SRA01/04.

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Selenite cataracts: activation of endoplasmic reticulum stress and loss of Nrf2/Keap1-dependent stress protection

http://www.wikidata.org/entity/Q34940558

description

2014 nî lūn-bûn

@nan

2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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Selenite cataracts: activation ...... p1-dependent stress protection

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J.BBADIS.2014.06.028

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Biochimica et Biophysica Acta

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Selenite cataracts: activation ...... p1-dependent stress protection

@en

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Keshore R Bidasee

http://www.w3.org/2001/XMLSchema#string

Periyasamy Palsamy

http://www.w3.org/2001/XMLSchema#string

Toshimichi Shinohara

http://www.w3.org/2001/XMLSchema#string

P2860

Endoplasmic reticulum oxidoreductin 1-lbeta (ERO1-Lbeta), a human gene induced in the course of the unfolded protein response

Mutations in the RNA granule component TDRD7 cause cataract and glaucoma

An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12

Nrf2 is a direct PERK substrate and effector of PERK-dependent cell survival

Oxidative protein folding in eukaryotes: mechanisms and consequences

Stress granules and processing bodies are dynamically linked sites of mRNP remodeling

Aging and age-related diseases of the ocular lens and vitreous body

Functional and genomic analyses reveal an essential coordination between the unfolded protein response and ER-associated degradation

Identification of NRF2, a member of the NF-E2 family of transcription factors, as a substrate for caspase-3(-like) proteases

The antioxidant responsive element. Activation by oxidative stress and identification of the DNA consensus sequence required for functional activity

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scholarly article

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10.1016/J.BBADIS.2014.06.028

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9

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1842

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2014-07-02T00:00:00Z

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24997453

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endoplasmic reticulum

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4293018

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