about
Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson's, Huntington's, Alzheimer's, prions, bactericides, chemical toxicology and others as examplesMitochondrial Dysfunction and Disturbed Coherence: Gate to Cancer.Asbestos-induced cellular and molecular alteration of immunocompetent cells and their relationship with chronic inflammation and carcinogenesisCurrent investigations into the genotoxicity of zinc oxide and silica nanoparticles in mammalian models in vitro and in vivo: carcinogenic/genotoxic potential, relevant mechanisms and biomarkers, artifacts, and limitationsDistinct affinity of nuclear proteins to the surface of chrysotile and crocidoliteMitochondria-derived reactive intermediate species mediate asbestos-induced genotoxicity and oxidative stress-responsive signaling pathwaysLung cancer and interstitial lung diseases: a systematic reviewOccIDEAS: An Innovative Tool to Assess Past Asbestos Exposure in the Australian Mesothelioma RegistryRole of mutagenicity in asbestos fiber-induced carcinogenicity and other diseasesQuantification of short and long asbestos fibers to assess asbestos exposure: a review of fiber size toxicity.Fenton reaction induced cancer in wild type rats recapitulates genomic alterations observed in human cancerDiameter and rigidity of multiwalled carbon nanotubes are critical factors in mesothelial injury and carcinogenesishsa-miR-29c* is linked to the prognosis of malignant pleural mesothelioma.A novel method for efficient collection of normal mesothelial cells in vivoPathophysiology and biology of peritoneal carcinomatosis.Synergistic defensive function of raphides and protease through the needle effect.Clinicopathologic and survival characteristics of malignant pleural mesothelioma registered in hospital cancer registryDual preventive benefits of iron elimination by desferal in asbestos-induced mesothelial carcinogenesisAccelerated cell cycle progression of human regulatory T cell-like cell line caused by continuous exposure to asbestos fibers.Urokinase-type plasminogen activator receptor promotes proliferation and invasion with reduced cisplatin sensitivity in malignant mesothelioma.Asbestos induces reduction of tumor immunity.Asbestos and multi-walled carbon nanotubes generate distinct oxidative responses in inflammatory cells.Evaluation of two distinct methods to quantify the uptake of crocidolite fibers by mesothelial cells.Inflammatory Alteration of Human T Cells Exposed Continuously to Asbestos.Biophysical insights into cancer transformation and treatment.
P2860
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P2860
description
2009 nî lūn-bûn
@nan
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Mechanisms of asbestos-induced carcinogenesis.
@ast
Mechanisms of asbestos-induced carcinogenesis.
@en
Mechanisms of asbestos-induced carcinogenesis.
@nl
type
label
Mechanisms of asbestos-induced carcinogenesis.
@ast
Mechanisms of asbestos-induced carcinogenesis.
@en
Mechanisms of asbestos-induced carcinogenesis.
@nl
prefLabel
Mechanisms of asbestos-induced carcinogenesis.
@ast
Mechanisms of asbestos-induced carcinogenesis.
@en
Mechanisms of asbestos-induced carcinogenesis.
@nl
P1476
Mechanisms of asbestos-induced carcinogenesis
@en
P2093
Shinya Toyokuni
P577
2009-02-01T00:00:00Z