Identification of rocA, a positive regulator of covR expression in the group A streptococcus.
about
A genome-wide study of two-component signal transduction systems in eight newly sequenced mutans streptococci strainsRole of VltAB, an ABC transporter complex, in viologen tolerance in Streptococcus mutansRegulation of gbpC expression in Streptococcus mutans.CovR-controlled global regulation of gene expression in Streptococcus mutansTwo group A streptococcal peptide pheromones act through opposing Rgg regulators to control biofilm development.RscA, a member of the MDR1 family of transporters, is repressed by CovR and required for growth of Streptococcus pyogenes under heat stress.CovS inactivates CovR and is required for growth under conditions of general stress in Streptococcus pyogenesMoonlighting glutamate formiminotransferases can functionally replace 5-formyltetrahydrofolate cycloligase.Genome-wide identification of genes required for fitness of group A Streptococcus in human bloodRivR and the small RNA RivX: the missing links between the CovR regulatory cascade and the Mga regulon.A two-component covRS regulatory system regulates expression of fructosyltransferase and a novel extracellular carbohydrate in Streptococcus mutansIncrease in pilus islet 2-encoded pili among Streptococcus pneumoniae isolates, Atlanta, Georgia, USA.RocA truncation underpins hyper-encapsulation, carriage longevity and transmissibility of serotype M18 group A streptococci.Group A Streptococcus transcriptome dynamics during growth in human blood reveals bacterial adaptive and survival strategies.Multimerization of the Virulence-Enhancing Group A Streptococcus Transcription Factor RivR Is Required for Regulatory Activity.Regulatory rewiring confers serotype-specific hyper-virulence in the human pathogen group A StreptococcusInvolvement of sensor kinases in the stress tolerance response of Streptococcus mutans.Inactivation of the ciaH Gene in Streptococcus mutans diminishes mutacin production and competence development, alters sucrose-dependent biofilm formation, and reduces stress tolerance.Quinolinate salvage and insights for targeting NAD biosynthesis in group A streptococci.Regulation of cytotoxin expression by converging eukaryotic-type and two-component signalling mechanisms in Streptococcus agalactiae.Intergenic Variable-Number Tandem-Repeat Polymorphism Upstream of rocA Alters Toxin Production and Enhances Virulence in Streptococcus pyogenes3'-Phosphoadenosine-5'-phosphate phosphatase activity is required for superoxide stress tolerance in Streptococcus mutansNull Mutations of Group A Streptococcus Orphan Kinase RocA: Selection in Mouse Infection and Comparison with CovS Mutations in Alteration of In Vitro and In Vivo Protease SpeB Expression and Virulence.Insights into Streptococcus pyogenes pathogenesis from transcriptome studies.Quorum sensing in group A StreptococcusRegulation of the glucosyltransferase (gtfBC) operon by CovR in Streptococcus mutansRocA Is an Accessory Protein to the Virulence-Regulating CovRS Two-Component System in Group A Streptococcus.A conserved streptococcal membrane protein, LsrS, exhibits a receptor-like function for lantibiotics.Regulatory gene mutation: a driving force behind group a Streptococcus strain- and serotype-specific variation.Comparative Genomics of the Mucoid and Nonmucoid Strains of Streptococcus pyogenes, Isolated from the Same Patient with Streptococcal Meningitis.LiaS regulates virulence factor expression in Streptococcus mutans.Modulation of covR expression in Streptococcus mutans UA159.An extracelluar protease, SepM, generates functional competence-stimulating peptide in Streptococcus mutans UA159.Mutacins from Streptococcus mutans UA159 are active against multiple streptococcal species.The cryptic competence pathway in Streptococcus pyogenes is controlled by a peptide pheromone.CovR alleviates transcriptional silencing by a nucleoid-associated histone-like protein in Streptococcus mutans.Unraveling the regulatory network in Streptococcus pyogenes: the global response regulator CovR represses rivR directly.Reply to "A truncation in the regulator RocA underlies heightened capsule expression in serotype M3 group A streptococci".Identification of β-haemolysin-encoding genes in Streptococcus anginosus.Phosphatase activity of the control of virulence sensor kinase CovS is critical for the pathogenesis of group A streptococcus
P2860
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P2860
Identification of rocA, a positive regulator of covR expression in the group A streptococcus.
description
2003 nî lūn-bûn
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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2003年の論文
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2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
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name
Identification of rocA, a posi ...... in the group A streptococcus.
@ast
Identification of rocA, a posi ...... in the group A streptococcus.
@en
Identification of rocA, a posi ...... in the group A streptococcus.
@nl
type
label
Identification of rocA, a posi ...... in the group A streptococcus.
@ast
Identification of rocA, a posi ...... in the group A streptococcus.
@en
Identification of rocA, a posi ...... in the group A streptococcus.
@nl
prefLabel
Identification of rocA, a posi ...... in the group A streptococcus.
@ast
Identification of rocA, a posi ...... in the group A streptococcus.
@en
Identification of rocA, a posi ...... in the group A streptococcus.
@nl
P2860
P1476
Identification of rocA, a posi ...... in the group A streptococcus.
@en
P2093
Indranil Biswas
June R Scott
P2860
P304
P356
10.1128/JB.185.10.3081-3090.2003
P407
P577
2003-05-01T00:00:00Z