l-Arginine depletion blunts antitumor T-cell responses by inducing myeloid-derived suppressor cells. - Wikidata - wikimedia - TriplyDB

l-Arginine depletion blunts antitumor T-cell responses by inducing myeloid-derived suppressor cells.

l-Arginine depletion blunts antitumor T-cell responses by inducing myeloid-derived suppressor cells.

http://www.wikidata.org/entity/Q34979666

about

Novel drugs that target the metabolic reprogramming in renal cell cancer Myeloid-Derived Suppressor Cells: Critical Cells Driving Immune Suppression in the Tumor Microenvironment T cell metabolism drives immunity The innate and adaptive infiltrating immune systems as targets for breast cancer immunotherapy.L-Arginine supplementation inhibits the growth of breast cancer by enhancing innate and adaptive immune responses mediated by suppression of MDSCs in vivo.Monocytic myeloid-derived suppressor cells from females, but not males, alleviate CVB3-induced myocarditis by increasing regulatory and CD4(+)IL-10(+) T cells.Amino acid auxotrophy as a system of immunological control nodes.Radiation Therapy Induces Macrophages to Suppress T-Cell Responses Against Pancreatic Tumors in Mice Prioritization schema for immunotherapy clinical trials in glioblastoma.Reconstitution of T Cell Proliferation under Arginine Limitation: Activated Human T Cells Take Up Citrulline via L-Type Amino Acid Transporter 1 and Use It to Regenerate Arginine after Induction of Argininosuccinate Synthase Expression.Similarities in the Metabolic Reprogramming of Immune System and Endothelium Temperature induces significant changes in both glycolytic reserve and mitochondrial spare respiratory capacity in colorectal cancer cell lines.Molecular basis and current strategies of therapeutic arginine depletion for cancer.Metabolic regulation of immune responses: therapeutic opportunities.When Is It Appropriate to Use Arginine in Critical Illness?Parenteral or Enteral Arginine Supplementation Safety and Efficacy.Understanding the Intersections between Metabolism and Cancer Biology.Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity.Metabolic Hallmarks of Tumor and Immune Cells in the Tumor Microenvironment Energy metabolic pathways control the fate and function of myeloid immune cells.The interplay between metabolic remodeling and immune regulation in glioblastoma.Metabolic Interactions in the Tumor Microenvironment.Metabolism in Immune Cell Differentiation and Function.Granulocytic myeloid-derived suppressor cells suppress virus-specific CD8+ T cell responses during acute Friend retrovirus infection.Deep Immune Profiling of an Arginine-Enriched Nutritional Intervention in Patients Undergoing Surgery.LMP1-mediated glycolysis induces myeloid-derived suppressor cell expansion in nasopharyngeal carcinoma.IGFBP7 Deletion Promotes Hepatocellular Carcinoma.Dysfunctional Natural Killer Cells in the Aftermath of Cancer Surgery.PEGylated arginine deiminase can modulate tumor immune microenvironment by affecting immune checkpoint expression, decreasing regulatory T cell accumulation and inducing tumor T cell infiltration.Mesenchymal stromal cells promote B-cell lymphoma in lacrimal glands by inducing immunosuppressive microenvironment.The arginine metabolome in acute lymphoblastic leukemia can be targeted by the pegylated-recombinant arginase I BCT-100.The spectrum of T cell metabolism in health and disease.Putative predictors of efficacy for immune checkpoint inhibitors in non-small-cell lung cancer: facing the complexity of the immune system.Metabolic Barriers to T Cell Function in Tumors.Mesenchymal stromal cells enhance the suppressive effects ofmyeloid-derived suppressor cells of multiple myeloma.Impact of l-Arginine Metabolism on Immune Response and Anticancer Immunotherapy.Immune cell landscape in therapy-naïve squamous cell and adenocarcinomas of the lung.Arginase-1 expression in myeloid cells regulates S. aureus planktonic but not biofilm infection.Cancer Immunotherapy: A Focus on the Regulation of Immune Checkpoints.Immunosuppressive Role of Myeloid-Derived Suppressor Cells and Therapeutic Targeting in Lung Cancer.

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l-Arginine depletion blunts antitumor T-cell responses by inducing myeloid-derived suppressor cells.

http://www.wikidata.org/entity/Q34979666

description

2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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l-Arginine depletion blunts an ...... loid-derived suppressor cells.

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l-Arginine depletion blunts an ...... loid-derived suppressor cells.

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l-Arginine depletion blunts an ...... loid-derived suppressor cells.

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0008-5472.CAN-14-1491

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Cancer Research

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l-Arginine depletion blunts an ...... loid-derived suppressor cells.

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Amir A Al-Khami

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Augusto C Ochoa

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Claudia Hernandez

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Dorota D Wyczechowska

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Dulfary Sanchez-Pino

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Maria E Ramirez

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Matthew Fletcher

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Patrick Raber

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Paul Thevenot

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Paulo C Rodriguez

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P2860

Posttranscriptional control of T cell effector function by aerobic glycolysis

Fueling immunity: insights into metabolism and lymphocyte function

Coordinated regulation of myeloid cells by tumours

The GCN2 kinase biases feeding behavior to maintain amino acid homeostasis in omnivores

The eIF2 kinase GCN2 is essential for the murine immune system to adapt to amino acid deprivation by asparaginase

Pegylated arginase I: a potential therapeutic approach in T-ALL

The stress-response sensor chop regulates the function and accumulation of myeloid-derived suppressor cells in tumors

Functional polarization of tumour-associated macrophages by tumour-derived lactic acid.

Bone marrow myeloid-derived suppressor cells (MDSCs) inhibit graft-versus-host disease (GVHD) via an arginase-1-dependent mechanism that is up-regulated by interleukin-13

L-arginine deprivation regulates cyclin D3 mRNA stability in human T cells by controlling HuR expression.

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275-283

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10.1158/0008-5472.CAN-14-1491

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2

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75

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2014-11-18T00:00:00Z

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25406192

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4297565

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