Fnip1 regulates skeletal muscle fiber type specification, fatigue resistance, and susceptibility to muscular dystrophy.
about
Globally Optimized Targeted Mass Spectrometry: Reliable Metabolomics Analysis with Broad CoverageCombining NMR and LC/MS Using Backward Variable Elimination: Metabolomics Analysis of Colorectal Cancer, Polyps, and Healthy Controls.A new therapeutic effect of simvastatin revealed by functional improvement in muscular dystrophy.Combinatorial therapeutic activation with heparin and AICAR stimulates additive effects on utrophin A expression in dystrophic muscles.Chronic AMPK activation via loss of FLCN induces functional beige adipose tissue through PGC-1α/ERRα.Mutation of Fnip1 is associated with B-cell deficiency, cardiomyopathy, and elevated AMPK activity.Overexpression of miR-26b-5p regulates the cell cycle by targeting CCND2 in GC-2 cells under exposure to extremely low frequency electromagnetic fieldsDistribution of myosin heavy chain isoforms in muscular dystrophy: insights into disease pathology.Coupling of mitochondrial function and skeletal muscle fiber type by a miR-499/Fnip1/AMPK circuit.Nutrient-induced FNIP degradation by SCFβ-TRCP regulates FLCN complex localization and promotes renal cancer progressionBirt-Hogg-Dubé syndrome: Clinical and molecular aspects of recently identified kidney cancer syndrome.Control of B lymphocyte development and functions by the mTOR signaling pathways.Chimpanzee super strength and human skeletal muscle evolution.Poly(C)-binding protein 1 (Pcbp1) regulates skeletal muscle differentiation by modulating microRNA processing in myoblasts.Muscle-specific deletion of Prkaa1 enhances skeletal muscle lipid accumulation in mice fed a high-fat diet.FLCN: The causative gene for Birt-Hogg-Dubé syndrome.Changes in Muscle Metabolism are Associated with Phenotypic Variability in Golden Retriever Muscular Dystrophy.Loss of Fnip1 alters kidney developmental transcriptional program and synergizes with TSC1 loss to promote mTORC1 activation and renal cyst formation.Skeletal Muscle Metabolism in Duchenne and Becker Muscular Dystrophy-Implications for Therapies.Salivary metabolite profiling distinguishes patients with oral cavity squamous cell carcinoma from normal controls
P2860
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P2860
Fnip1 regulates skeletal muscle fiber type specification, fatigue resistance, and susceptibility to muscular dystrophy.
description
2014 nî lūn-bûn
@nan
2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
name
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@ast
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@en
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@nl
type
label
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@ast
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@en
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@nl
prefLabel
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@ast
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@en
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@nl
P2093
P2860
P356
P1476
Fnip1 regulates skeletal muscl ...... ibility to muscular dystrophy.
@en
P2093
Brian M Iritani
Daciana Margineantu
Daniel Raftery
Danijel Djukovic
David M Hockenbery
Dinesh K Hirenallur-S
Glen B Banks
H Denny Liggitt
Jacky Chan
P2860
P304
P356
10.1073/PNAS.1413021112
P407
P577
2014-12-29T00:00:00Z