miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b. - Wikidata - wikimedia - TriplyDB

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

http://www.wikidata.org/entity/Q34986314

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Identification of Differentially Expressed MicroRNAs in Osteosarcoma MicroRNA-210: a unique and pleiotropic hypoxamir MiR-210--micromanager of the hypoxia pathway An integrated approach for experimental target identification of hypoxia-induced miR-210 The Regulatory Roles of MicroRNAs in Bone Remodeling and Perspectives as Biomarkers in Osteoporosis TGF-β/BMP signaling and other molecular events: regulation of osteoblastogenesis and bone formation The role of microRNAs in bone remodeling The Role of MicroRNAs in Diabetic Complications-Special Emphasis on Wound Healing Vitamin D and microRNAs in bone OxymiRs in cutaneous development, wound repair and regeneration Regulation of gene expression in osteoblasts.MicroRNAs regulate bone development and regeneration miR-210: More than a silent player in hypoxia A Runx2/miR-3960/miR-2861 regulatory feedback loop during mouse osteoblast differentiation MiR-101 Targets the EZH2/Wnt/β-Catenin the Pathway to Promote the Osteogenic Differentiation of Human Bone Marrow-Derived Mesenchymal Stem Cells.Identification and characterization of microRNAs by high through-put sequencing in mesenchymal stem cells and bone tissue from mice of age-related osteoporosis.MicroRNA-146a regulates human foetal femur derived skeletal stem cell differentiation by down-regulating SMAD2 and SMAD3.Multiple functions of hypoxia-regulated miR-210 in cancer.miR-29 modulates Wnt signaling in human osteoblasts through a positive feedback loop.HypoxamiR regulation and function in ischemic cardiovascular diseases.miR-764-5p promotes osteoblast differentiation through inhibition of CHIP/STUB1 expression.Expression of BMP-2 and Ets1 in BMP-2-stimulated mouse pre-osteoblast differentiation is regulated by microRNA-370.Functional screening for miRNAs targeting Smad4 identified miR-199a as a negative regulator of TGF-β signalling pathway.Serum microRNA is a promising biomarker for osteogenesis imperfecta MicroRNAs involved in bone formation.A microRNA expression signature of osteoclastogenesis microRNA-21 mediates stretch-induced osteogenic differentiation in human periodontal ligament stem cells Elevated expression of miR-210 predicts poor survival of cancer patients: a systematic review and meta-analysis.Photoactivated miR-148b-nanoparticle conjugates improve closure of critical size mouse calvarial defects Gene expression and hypoxia in breast cancer.Abnormalities associated with progressive aortic vascular dysfunction in chronic kidney disease.miR-210: fine-tuning the hypoxic response.Pathogenesis of glucocorticoid-induced avascular necrosis: A microarray analysis of gene expression in vitro Do epigenetic marks govern bone mass and homeostasis?Mesenchymal Stem/Stromal Cells under Stress Increase Osteosarcoma Migration and Apoptosis Resistance via Extracellular Vesicle Mediated Communication.Transforming growth factor-β, bioenergetics, and mitochondria in renal disease MicroRNAs' Involvement in Osteoarthritis and the Prospects for Treatments.miR-210 has an antiapoptotic effect in pulmonary artery smooth muscle cells during hypoxia Hypoxia-inducible factor 1-α induces miR-210 in normoxic differentiating myoblasts.Hypoxia-inducible miR-210 contributes to preeclampsia via targeting thrombospondin type I domain containing 7A.

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miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

http://www.wikidata.org/entity/Q34986314

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@ast

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@en

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@nl

type

label

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@ast

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@en

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@nl

prefLabel

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@ast

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@en

miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@nl

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J.FEBSLET.2009.06.006

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miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b.

@en

P2093

Hideo Tashiro

http://www.w3.org/2001/XMLSchema#string

Ken Yagi

http://www.w3.org/2001/XMLSchema#string

Takenobu Katagiri

http://www.w3.org/2001/XMLSchema#string

Tatsuo Suda

http://www.w3.org/2001/XMLSchema#string

Tomoyuki Amemiya

http://www.w3.org/2001/XMLSchema#string

Toru Fukuda

http://www.w3.org/2001/XMLSchema#string

Yasumitsu Kondoh

http://www.w3.org/2001/XMLSchema#string

Yasushi Okazaki

http://www.w3.org/2001/XMLSchema#string

Yoshimi Tokuzawa

http://www.w3.org/2001/XMLSchema#string

Yosuke Mizuno

http://www.w3.org/2001/XMLSchema#string

P2860

Endogenous TGF-beta signaling suppresses maturation of osteoblastic mesenchymal cells

miR-210 links hypoxia with cell cycle regulation and is deleted in human epithelial ovarian cancer

A microRNA signature for a BMP2-induced osteoblast lineage commitment program

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

The tumor suppressor microRNA let-7 represses the HMGA2 oncogene.

Prediction of mammalian microRNA targets

Combinatorial microRNA target predictions

c-Myc-regulated microRNAs modulate E2F1 expression

let-7 microRNA functions as a potential growth suppressor in human colon cancer cells

The let-7 microRNA represses cell proliferation pathways in human cells

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10.1016/J.FEBSLET.2009.06.006

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13

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26286166

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19520079

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