Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
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The role of immunoglobulin superfamily cell adhesion molecules in cancer metastasisNon-thermal atmospheric pressure plasmas as a novel candidate for preventive therapy of melanoma.Decreased expression levels of cell cycle regulators and matrix metalloproteinases in melanoma from RET-transgenic mice by single irradiation of non-equilibrium atmospheric pressure plasmas.LIF negatively regulates tumour-suppressor p53 through Stat3/ID1/MDM2 in colorectal cancers.Cytoplasmic accumulation of NCoR in malignant melanoma: consequences of altered gene repression and prognostic significanceHuman fetal skeletal muscle contains a myogenic side population that expresses the melanoma cell-adhesion molecule.Nanobodies against surface biomarkers enable the analysis of tumor genetic heterogeneity in uveal melanoma patient-derived xenografts.Downregulation of Melanoma Cell Adhesion Molecule (MCAM/CD146) Accelerates Cellular Senescence in Human Umbilical Cord Blood-Derived Mesenchymal Stem Cells.Id Proteins Contribute to Tumor Development and Metastatic Colonization in a Model of Bladder Carcinogenesis.Masked selection: a straightforward and flexible approach for the selection of binders against specific epitopes and differentially expressed proteins by phage display.The activating transcription factor 3 protein suppresses the oncogenic function of mutant p53 proteins.Molecular networks in melanoma invasion and metastasis.Melanoma biomolecules: independently identified but functionally intertwined.Why is melanoma so metastatic?Ectopic expression of MCAM/MUC18 increases in vitro motility and invasiveness, but decreases in vivo tumorigenesis and metastasis of a mouse melanoma K1735-9 subline in a syngeneic mouse model.The multifaceted role of CD146/MCAM in the promotion of melanoma progression.The Id-protein family in developmental and cancer-associated pathways.Microarray analyses reveal liver metastasis-related genes in metastatic colorectal cancer cell model.MCAM contributes to the establishment of cell autonomous polarity in myogenic and chondrogenic differentiation.MCAM expression is associated with poor prognosis in non-small cell lung cancer.
P2860
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P2860
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
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2011 nî lūn-bûn
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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2011 թվականի ապրիլին հրատարակված գիտական հոդված
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2011年の論文
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2011年論文
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2011年論文
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2011年論文
@zh-hk
2011年論文
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2011年論文
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2011年论文
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name
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@ast
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@en
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@nl
type
label
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@ast
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@en
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@nl
prefLabel
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@ast
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@en
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@nl
P2093
P2860
P1433
P1476
Expression of Id-1 is regulated by MCAM/MUC18: a missing link in melanoma progression
@en
P2093
Andrey S Dobroff
Gabriel J Villares
Maya Zigler
Menashe Bar-Eli
Ran Friedman
Renduo Song
Russell R Braeuer
Takafumi Kamiya
P2860
P304
P356
10.1158/0008-5472.CAN-10-3555
P407
P577
2011-04-05T00:00:00Z