c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype. - Wikidata - wikimedia - TriplyDB

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

http://www.wikidata.org/entity/Q34988841

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Gene Therapy in Cardiac Surgery: Clinical Trials, Challenges, and Perspectives Stromal derived factor-1 mediates the lung regenerative effects of mesenchymal stem cells in a rodent model of bronchopulmonary dysplasia.Expression profiling of lymph nodes in tuberculosis patients reveal inflammatory milieu at site of infection.Endothelial Mesenchymal Transition: Comparative Analysis of Different Induction Methods.IL-3R-alpha blockade inhibits tumor endothelial cell-derived extracellular vesicle (EV)-mediated vessel formation by targeting the β-catenin pathway.A review of the multifunctionality of angiopoietin-like 4 in eye disease A systems biology network analysis of nutri(epi)genomic changes in endothelial cells exposed to epicatechin metabolites

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c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

http://www.wikidata.org/entity/Q34988841

description

2013 nî lūn-bûn

@nan

2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@ast

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@en

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@nl

type

label

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

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c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@en

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@nl

prefLabel

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@ast

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@en

c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

@nl

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JOURNAL.PONE.0073146

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c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.

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Claudia O Rodrigues

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Francisco Y Macedo

http://www.w3.org/2001/XMLSchema#string

Gordana Simovic

http://www.w3.org/2001/XMLSchema#string

Nithya Bhagavatula

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Ricardo A Fock

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Victoria Florea

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P2860

VEGI, a novel cytokine of the tumor necrosis factor family, is an angiogenesis inhibitor that suppresses the growth of colon carcinomas in vivo

Obesity and cancer risk: recent review and evidence

Angiopoietin-like 4 is a proangiogenic factor produced during ischemia and in conventional renal cell carcinoma

Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins

Cancer-related inflammation

A role for smad6 in development and homeostasis of the cardiovascular system

Proliferation, cell cycle and apoptosis in cancer

Repression of p15INK4b expression by Myc through association with Miz-1

Emerging roles of angiopoietin-like 4 in human cancer

c-Myc is a universal amplifier of expressed genes in lymphocytes and embryonic stem cells

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24039874

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