The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling - Wikidata - wikimedia - TriplyDB

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

http://www.wikidata.org/entity/Q34989767

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Neutrophil microvesicles resolve gout by inhibiting C5a-mediated priming of the inflammasome How Mouse Macrophages Sense What Is Going On.Recent advances in mechanisms and treatments of airway remodeling in asthma: a message from the bench side to the clinic Impaired induction of allergic lung inflammation by Alternaria alternata mutant MAPK homologue Fus3 Functional effects of TGF-β1 on mesenchymal stem cell mobilization in cockroach allergen-induced asthma Systems biology approaches to understanding Epithelial Mesenchymal Transition (EMT) in mucosal remodeling and signaling in asthma Identification of genes expressed by human airway eosinophils after an in vivo allergen challenge.Cloning, expression, and characterization of TNFSF14 (LIGHT) gene in mefugu, Takifugu obscurus.Licensed human natural killer cells aid dendritic cell maturation via TNFSF14/LIGHT MicroRNA regulate immune pathways in T-cells in multiple sclerosis (MS).Suppressive effect of insulin on the gene expression and plasma concentrations of mediators of asthmatic inflammation.Lymphotoxin β receptor signaling induces IL-8 production in human bronchial epithelial cells.TNF Superfamily Networks: bidirectional and interference pathways of the herpesvirus entry mediator (TNFSF14)Donor B-cell alloantibody deposition and germinal center formation are required for the development of murine chronic GVHD and bronchiolitis obliterans Alternaria induces STAT6-dependent acute airway eosinophilia and epithelial FIZZ1 expression that promotes airway fibrosis and epithelial thickness.TNF superfamily in inflammatory disease: translating basic insights.The Tumor Necrosis Factor Superfamily Molecule LIGHT Promotes Keratinocyte Activity and Skin Fibrosis.Tumor necrosis factor superfamily 14 (LIGHT) controls thymic stromal lymphopoietin to drive pulmonary fibrosis.The signaling networks of the herpesvirus entry mediator (TNFRSF14) in immune regulation STAT6 regulates natural helper cell proliferation during lung inflammation initiated by Alternaria.Improvement in efficacy of DNA vaccine encoding HIV-1 Vif by LIGHT gene adjuvant HVEM: An unusual TNF receptor family member important for mucosal innate immune responses to microbes.Airway Hyperresponsiveness and Inflammation: Causation, Correlation, or No Relation?Fstl1 Promotes Asthmatic Airway Remodeling by Inducing Oncostatin M.Clinical targeting of the TNF and TNFR superfamilies Programmed cell death ligand 2 regulates TH9 differentiation and induction of chronic airway hyperreactivity The TNF Receptor Superfamily in Co-stimulating and Co-inhibitory Responses Virus-like particles presenting interleukin-33 molecules: immunization characteristics and potentials of blockingIL-33/ST2 pathway in allergic airway inflammation Immunotherapeutic targeting of LIGHT/LTβR/HVEM pathway fully recapitulates the reduced cytotoxic phenotype of LIGHT-deficient T cells.A TNFRSF14-FcɛRI-mast cell pathway contributes to development of multiple features of asthma pathology in mice.Innate type 2 response to Alternaria extract enhances ryegrass-induced lung inflammation.p38 MAPK inhibitors, IKK2 inhibitors, and TNFα inhibitors in COPD.Cross-roads in the lung: immune cells and tissue interactions as determinants of allergic asthma.The airway epithelium in asthma.LIGHT/HVEM/LTβR interaction as a target for the modulation of the allogeneic immune response in transplantation.T cell co-stimulatory molecules: a co-conspirator in the pathogenesis of eosinophilic esophagitis?Genome-wide association studies of atopic dermatitis.Differential gene expression profiles of peripheral blood mononuclear cells in childhood asthma.The Genetics and Epigenetics of Atopic Dermatitis-Filaggrin and Other Polymorphisms.Effect of tumor necrosis factor family member LIGHT (TNFSF14) on the activation of basophils and eosinophils interacting with bronchial epithelial cells.

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The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

http://www.wikidata.org/entity/Q34989767

description

2011 nî lūn-bûn

@nan

2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

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The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

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The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

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type

label

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

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The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

@en

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

@nl

prefLabel

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

@ast

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

@en

The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

@nl

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Nature Medicine

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The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling

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Bruce L Zuraw

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Carl F Ware

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David H Broide

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Heonsik Choi

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Jae Youn Cho

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Michael Croft

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Naseem Khorram

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Paula S Norris

http://www.w3.org/2001/XMLSchema#string

Pejman Soroosh

http://www.w3.org/2001/XMLSchema#string

Peter Rosenthal

http://www.w3.org/2001/XMLSchema#string

P2860

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

Detection and quantitation of eosinophils in the murine respiratory tract by flow cytometry.

Treatment of experimental (Trinitrobenzene sulfonic acid) colitis by intranasal administration of transforming growth factor (TGF)-beta1 plasmid: TGF-beta1-mediated suppression of T helper cell type 1 response occurs by interleukin (IL)-10 induction

CysLT1 receptor upregulation by TGF-beta and IL-13 is associated with bronchial smooth muscle cell proliferation in response to LTD4

Expression of three members of the TNF-R family of receptors (4-1BB, lymphotoxin-beta receptor, and Fas) in human lung

IL-13-induced proliferation of airway epithelial cells: mediation by intracellular growth factor mobilization and ADAM17

House dust mite allergen induces asthma via Toll-like receptor 4 triggering of airway structural cells.

Neutralisation of interleukin-13 in mice prevents airway pathology caused by chronic exposure to house dust mite.

Inducible expression of inflammatory chemokines in respiratory syncytial virus-infected mice: role of MIP-1alpha in lung pathology.

Analyses of asthma severity phenotypes and inflammatory proteins in subjects stratified by sputum granulocytes

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596-603

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scholarly article

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10.1038/NM.2356

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P433

5

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17

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Taylor A Doherty

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2011-04-17T00:00:00Z

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51057116

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21499267

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3097134

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