The optic nerve head is the site of axonal transport disruption, axonal cytoskeleton damage and putative axonal regeneration failure in a rat model of glaucoma - Wikidata - wikimedia - TriplyDB

The optic nerve head is the site of axonal transport disruption, axonal cytoskeleton damage and putative axonal regeneration failure in a rat model of glaucoma

The optic nerve head is the site of axonal transport disruption, axonal cytoskeleton damage and putative axonal regeneration failure in a rat model of glaucoma

http://www.wikidata.org/entity/Q34995800

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Critical pathogenic events underlying progression of neurodegeneration in glaucoma In vivo imaging methods to assess glaucomatous optic neuropathy Ocular expression and distribution of products of the POAG-associated chromosome 9p21 gene region Influence of transient intraocular pressure elevation during laser in situ keratomileusis on rabbit retina thickness.Metrics of the normal anterior sclera: imaging with optical coherence tomography Quantification of retrograde axonal transport in the rat optic nerve by fluorogold spectrometry Gene Expression Profiling of the Optic Nerve Head of Patients with Primary Open-Angle Glaucoma.Localization of a wide-ranging panel of antigens in the rat retina by immunohistochemistry: comparison of Davidson's solution and formalin as fixatives Astrocyte Reactivity: A Biomarker for Retinal Ganglion Cell Health in Retinal Neurodegeneration Estimation of axon counts in a rat model of glaucoma: comparison of fixed-pattern sampling with targeted sampling.Definition of glaucoma: clinical and experimental concepts.Wavelength-dependent change of retinal nerve fiber layer reflectance in glaucomatous retinas.Comparison of longitudinal in vivo measurements of retinal nerve fiber layer thickness and retinal ganglion cell density after optic nerve transection in rat.Imaging axonal transport in the rat visual pathway.Comparison of retinal nerve fiber layer thickness in vivo and axonal transport after chronic intraocular pressure elevation in young versus older rats.Does optic nerve head surface topography change prior to loss of retinal nerve fiber layer thickness: a test of the site of injury hypothesis in experimental glaucoma.Neuroprotective effects of transcription factor Brn3b in an ocular hypertension rat model of glaucoma Axonal transport declines with age in two distinct phases separated by a period of relative stability.Evaluation of retinal nerve fiber layer thickness and axonal transport 1 and 2 weeks after 8 hours of acute intraocular pressure elevation in rats.Dark rearing maintains tyrosine hydroxylase expression in retinal amacrine cells following optic nerve transection Distinct terminal and cell body mechanisms in the nociceptor mediate hyperalgesic priming Expression of inducible heat shock proteins Hsp27 and Hsp70 in the visual pathway of rats subjected to various models of retinal ganglion cell injury.Effect of suction on macular thickness and retinal nerve fiber layer thickness during LASIK used femtosecond laser and Moria M2 microkeratome Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucoma β-III-Tubulin: a reliable marker for retinal ganglion cell labeling in experimental models of glaucoma.Caffeine administration prevents retinal neuroinflammation and loss of retinal ganglion cells in an animal model of glaucoma.Distribution pattern of axonal cytoskeleton proteins in the human optic nerve head.Optic nerve astrocyte reactivity protects function in experimental glaucoma and other nerve injuries.Retina-on-a-chip: a microfluidic platform for point access signaling studies Failure of axonal transport induces a spatially coincident increase in astrocyte BDNF prior to synapse loss in a central target.Retrograde signaling in the optic nerve is necessary for electrical responsiveness of retinal ganglion cells.Early Cytoskeletal Protein Modifications Precede Overt Structural Degeneration in the DBA/2J Mouse Model of Glaucoma.Loss of Fractalkine Signaling Exacerbates Axon Transport Dysfunction in a Chronic Model of Glaucoma.Intrinsic axonal degeneration pathways are critical for glaucomatous damage.Review: Axon pathology in age-related neurodegenerative disorders.Glaucoma and optic nerve repair.Mini-Review: Impaired Axonal Transport and Glaucoma.Red light of the visual spectrum attenuates cell death in culture and retinal ganglion cell death in situ.BAX to basics: How the BCL2 gene family controls the death of retinal ganglion cells.Cross-linked actin networks (CLANs) in glaucoma.

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The optic nerve head is the site of axonal transport disruption, axonal cytoskeleton damage and putative axonal regeneration failure in a rat model of glaucoma

http://www.wikidata.org/entity/Q34995800

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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The optic nerve head is the si ...... ure in a rat model of glaucoma

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Acta Neuropathologica

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The optic nerve head is the si ...... ure in a rat model of glaucoma

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Glyn Chidlow

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John P M Wood

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Robert J Casson

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A new mathematical model for relative quantification in real-time RT-PCR

Brain-derived neurotrophic factor is an anterograde survival factor in the rat visual system

Translimbal laser photocoagulation to the trabecular meshwork as a model of glaucoma in rats.

The expression of heat shock protein 27 in retinal ganglion and glial cells in a rat glaucoma model.

Increase in dephosphorylation of the heavy neurofilament subunit in the monkey chronic glaucoma model.

Progressive ganglion cell loss and optic nerve degeneration in DBA/2J mice is variable and asymmetric

Ocular hypertension impairs optic nerve axonal transport leading to progressive retinal ganglion cell degeneration.

Microglial activation in the visual pathway in experimental glaucoma: spatiotemporal characterization and correlation with axonal injury.

Distal axonopathy with structural persistence in glaucomatous neurodegeneration.

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Andreas Ebneter

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