Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA. - Wikidata - wikimedia - TriplyDB

Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

http://www.wikidata.org/entity/Q35002489

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Comprehensive identification of cell cycle-regulated genes of the yeast Saccharomyces cerevisiae by microarray hybridization Unequal division in Saccharomyces cerevisiae and its implications for the control of cell division RPC53 encodes a subunit of Saccharomyces cerevisiae RNA polymerase C (III) whose inactivation leads to a predominantly G1 arrest A systematic analysis of cell cycle regulators in yeast reveals that most factors act independently of cell size to control initiation of division A survey of essential gene function in the yeast cell division cycle Methionine inhibits autophagy and promotes growth by inducing the SAM-responsive methylation of PP2A Cell size control in yeast Protein synthesis requirements for nuclear division, cytokinesis, and cell separation in Saccharomyces cerevisiae Ribosome biogenesis is sensed at the Start cell cycle checkpoint.Sulfur metabolism actively promotes initiation of cell division in yeast.Cell-size dependent progression of the cell cycle creates homeostasis and flexibility of plant cell size Growth-limiting intracellular metabolites in yeast growing under diverse nutrient limitations.Growth-independent regulation of CLN3 mRNA levels by nutrients in Saccharomyces cerevisiae.Cytocidal amino acid starvation of Saccharomyces cerevisiae and Candida albicans acetolactate synthase (ilv2{Delta}) mutants is influenced by the carbon source and rapamycin.Differential growth rates of Candida utilis mother and daughter cells under phased cultivation.Robust metabolic responses to varied carbon sources in natural and laboratory strains of Saccharomyces cerevisiae Reserve carbohydrate metabolism in Saccharomyces cerevisiae: responses to nutrient limitation Degradation of the transcription factor Gcn4 requires the kinase Pho85 and the SCF(CDC4) ubiquitin-ligase complex Metabolic status rather than cell cycle signals control quiescence entry and exit SCF(Met30)-mediated control of the transcriptional activator Met4 is required for the G(1)-S transition 31P NMR studies of intracellular pH and phosphate metabolism during cell division cycle of Saccharomyces cerevisiae.Nature of the G1 phase of the yeast Saccharomyces cerevisiae.Survival of starving yeast is correlated with oxidative stress response and nonrespiratory mitochondrial function.The Levels of a Universally Conserved tRNA Modification Regulate Cell Growth.Cell cycle synchronization by nutrient modulation Combinatorial control of diverse metabolic and physiological functions by transcriptional regulators of the yeast sulfur assimilation pathway.Glucose, nitrogen, and phosphate repletion in Saccharomyces cerevisiae: common transcriptional responses to different nutrient signals.Regulation of mating in the cell cycle of Saccharomyces cerevisiae Cell division from a genetic perspective.Coordination of growth rate, cell cycle, stress response, and metabolic activity in yeast.Addition of basic amino acids prevents G-1 arrest of nitrogen-starved cultures of Saccharomyces cerevisiae.Control of vacuole permeability and protein degradation by the cell cycle arrest signal in Saccharomyces cerevisiae Comparative functional genomic screens of three yeast deletion collections reveal unexpected effects of genotype in response to diverse stress.Growth and cell division during nitrogen starvation of the yeast Saccharomyces cerevisiae.Methionyl-transfer ribonucleic acid deficiency during G1 arrest of Saccharomyces cerevisiae.Influence of genotype and nutrition on survival and metabolism of starving yeast Mechanisms of gene regulation in the general control of amino acid biosynthesis in Saccharomyces cerevisiae.Five conditions commonly used to down-regulate tor complex 1 generate different physiological situations exhibiting distinct requirements and outcomes.Kinetic partitioning during de novo septin filament assembly creates a critical G1 "window of opportunity" for mutant septin function.Glucose induces cAMP-independent growth-related changes in stationary-phase cells of Saccharomyces cerevisiae.

P2860

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P2860

Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

http://www.wikidata.org/entity/Q35002489

description

1976 nî lūn-bûn

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1976 թուականի Մայիսին հրատարակուած գիտական յօդուած

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1976 թվականի մայիսին հրատարակված գիտական հոդված

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1976年の論文

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1976年論文

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1976年論文

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1976年論文

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1976年論文

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1976年論文

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1976年论文

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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type

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Proceedings of the National Academy of Sciences of the United States of America

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Control of cell division in Saccharomyces cerevisiae by methionyl-tRNA.

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Hartwell LH

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A restriction point for control of normal animal cell proliferation

Methionine biosynthesis in Saccharomyces cerevisiae. I. Genetical analysis of auxotrophic mutants.

Duplication of spindle plaques and integration of the yeast cell cycle

Behavior of spindles and spindle plaques in the cell cycle and conjugation of Saccharomyces cerevisiae

Genetic control of the cell division cycle in yeast

A mutant of yeast with a defective methionyl-tRNA synthetase.

Sequential replication of Bacillus subtilis chromosome. I. Comparison of marker frequencies in exponential and stationary growth phases

Induction of growth in resting fibroblastic cell cultures by Ca++.

Central role for magnesium in coordinate control of metabolism and growth in animal cells.

Transport changes rapidly initiated by serum addition to "contact inhibited" 3T3 cells.

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