c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity - Wikidata - wikimedia - TriplyDB

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

http://www.wikidata.org/entity/Q35005009

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Critical role of c-Jun overexpression in liver metastasis of human breast cancer xenograft model Roles of the ERK, JNK/AP-1/cyclin D1-CDK4 pathway in silica-induced cell cycle changes in human embryo lung fibroblast cells Lack of p21 expression links cell cycle control and appendage regeneration in mice Interactions between Myc and Mediators of Inflammation in Chronic Liver Diseases Altered expression of cell cycle and apoptotic proteins in chronic hepatitis C virus infection The role of p21 in regulating mammalian regeneration Molecular mechanisms of liver injury and hepatocarcinogenesis: focusing on the role of stress-activated MAPK A liver full of JNK: signaling in regulation of cell function and disease pathogenesis, and clinical approaches Impaired liver regeneration in Nrf2 knockout mice: role of ROS-mediated insulin/IGF-1 resistance Induction of p53-dependent p21 limits proliferative activity of rat hepatocytes in the presence of hepatocyte growth factor Global gene profiling of spontaneous hepatocellular carcinoma in B6C3F1 mice: similarities in the molecular landscape with human liver cancer Platelets in liver disease, cancer and regeneration Fibroblast growth factor 15 deficiency impairs liver regeneration in mice.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Tob1 is a constitutively expressed repressor of liver regeneration Progenitor-derived hepatocellular carcinoma model in the rat.Portal vein ligation accelerates tumor growth in ligated, but not contralateral lobes Cascades of transcription regulation during liver regeneration.Mammary gland selective excision of c-jun identifies its role in mRNA splicing.Targeted disruption of Mapk14 (p38MAPKalpha) in granulosa cells and cumulus cells causes cell-specific changes in gene expression profiles that rescue COC expansion and maintain fertility.Identification of microRNAs during rat liver regeneration after partial hepatectomy and modulation by ursodeoxycholic acid Hepatocyte proliferation during liver regeneration is impaired in mice with methionine diet-induced hyperhomocysteinemia.The transcription factor c-Jun protects against liver damage following activated β-Catenin signaling.Specific activin receptor-like kinase 3 inhibitors enhance liver regeneration Jun is required in Isl1-expressing progenitor cells for cardiovascular development The role of JNK in the development of hepatocellular carcinoma.Acquisition of an immunosuppressive protumorigenic macrophage phenotype depending on c-Jun phosphorylation.p53 Contributes to Differentiating Gene Expression Following Exposure to Acetaminophen and Its Less Hepatotoxic Regioisomer Both In Vitro and In Vivo.Reparative inflammation takes charge of tissue regeneration.Pregnane X Receptor-Humanized Mice Recapitulate Gender Differences in Ethanol Metabolism but Not Hepatotoxicity Oncostatin M gene therapy attenuates liver damage induced by dimethylnitrosamine in rats A novel comparative pattern analysis approach identifies chronic alcohol mediated dysregulation of transcriptomic dynamics during liver regeneration The EGF receptor is required for efficient liver regeneration Hepatocyte survival in acute hepatitis is due to c-Jun/AP-1-dependent expression of inducible nitric oxide synthase Differentiation-induced skin cancer suppression by FOS, p53, and TACE/ADAM17.WD-repeat protein WDR13 is a novel transcriptional regulator of c-Jun and modulates intestinal homeostasis in mice.Annexin A2 silencing induces G2 arrest of non-small cell lung cancer cells through p53-dependent and -independent mechanisms.Promoter-distal RNA polymerase II binding discriminates active from inactive CCAAT/ enhancer-binding protein beta binding sites Glutathione transferases P1/P2 regulate the timing of signaling pathway activations and cell cycle progression during mouse liver regeneration Combined CSL and p53 downregulation promotes cancer-associated fibroblast activation

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P2860

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

http://www.wikidata.org/entity/Q35005009

description

2006 nî lūn-bûn

@nan

2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2006年の論文

@ja

2006年論文

@yue

2006年論文

@zh-hant

2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

name

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@ast

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@en

type

label

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@ast

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@en

prefLabel

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@ast

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@en

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Genes & Development

P1476

c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity

@en

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Ewa Stepniak

http://www.w3.org/2001/XMLSchema#string

Grzegorz Sumara

http://www.w3.org/2001/XMLSchema#string

Izabela Sumara

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Lijian Hui

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Martina Rath

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P2860

p53-inducible wip1 phosphatase mediates a negative feedback regulation of p38 MAPK-p53 signaling in response to UV radiation

Control of cell cycle progression by c-Jun is p53 dependent

IFI16 as a negative regulator in the regulation of p53 and p21(Waf1)

Liver tumor development. c-Jun antagonizes the proapoptotic activity of p53

Functions of c-Jun in liver and heart development

CCAAT enhancer- binding protein beta is required for normal hepatocyte proliferation in mice after partial hepatectomy

JunB can substitute for Jun in mouse development and cell proliferation

Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

Inducible gene targeting in mice

Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control

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2306-2314

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10.1101/GAD.390506

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16

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20

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6875889

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16912279

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1553212

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