Is oxidative damage the fundamental pathogenic mechanism of Alzheimer's and other neurodegenerative diseases?
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Why nutraceuticals do not prevent or treat Alzheimer's diseaseRelevance of the glutathione system in temporal lobe epilepsy: evidence in human and experimental modelsAlzheimer's disease pathologic cascades: who comes first, what drives whatCritical evaluation of ayurvedic plants for stimulating intrinsic antioxidant response.Novel 5' untranslated region directed blockers of iron-regulatory protein-1 dependent amyloid precursor protein translation: implications for down syndrome and Alzheimer's diseaseRole of oxidative stress in epileptic seizures.High fat diet increases hippocampal oxidative stress and cognitive impairment in aged mice: implications for decreased Nrf2 signalingThe role of iron in brain ageing and neurodegenerative disorders.N-acetyl aspartate concentration in the anterior cingulate cortex in patients with schizophrenia: a study of clinical and neuropsychological correlates and preliminary exploration of cognitive behaviour therapy effectsRegion-specific effects on brain metabolites of hypoxia and hyperoxia overlaid on cerebral ischemia in young and old rats: a quantitative proton magnetic resonance spectroscopy study.Increased iron and free radical generation in preclinical Alzheimer disease and mild cognitive impairment.Environmental experience modulates ischemia-induced amyloidogenesis and enhances functional recovery.Biomarkers of oxidative damage and inflammation in Alzheimer's disease.Antioxidant therapy in Alzheimer's disease: theory and practice.Biomarkers of Alzheimer's disease risk in peripheral tissues; focus on buccal cells.Synergy between the C2 allele of transferrin and the C282Y allele of the haemochromatosis gene (HFE) as risk factors for developing Alzheimer's disease.A quantitative method to monitor reactive oxygen species production by electron paramagnetic resonance in physiological and pathological conditions.Autophagy failure in Alzheimer's disease--locating the primary defect.Oxidative Stress and its Implications for Future Treatments and Management of Alzheimer DiseaseOxidative stress in neurodegenerationAlzheimer's disease is not "brain aging": neuropathological, genetic, and epidemiological human studies.Protective effects of walnut extract against amyloid beta peptide-induced cell death and oxidative stress in PC12 cells.CR6-interacting factor 1 is a key regulator in Aβ-induced mitochondrial disruption and pathogenesis of Alzheimer's disease.Mitochondrial DNA deletions and differential mitochondrial DNA content in Rhesus monkeys: implications for agingOxidative stress in erythrocytes from patients with rheumatoid arthritisMitochondria as a therapeutic target for aging and neurodegenerative diseases.Cortical iron regulation and inflammatory response in Alzheimer's disease and APPSWE/PS1ΔE9 mice: a histological perspectiveMitochondria and cell bioenergetics: increasingly recognized components and a possible etiologic cause of Alzheimer's disease.Neuroprotection and neurodegeneration in Alzheimer's disease: role of cardiovascular disease risk factors, implications for dementia rates, and prevention with aerobic exercise in african americans.Role and treatment of mitochondrial DNA-related mitochondrial dysfunction in sporadic neurodegenerative diseases.Mitochondrial- and endoplasmic reticulum-associated oxidative stress in Alzheimer's disease: from pathogenesis to biomarkers.Reducing mitochondrial decay with mitochondrial nutrients to delay and treat cognitive dysfunction, Alzheimer's disease, and Parkinson's disease.Tau Hyperphosphorylation and Oxidative Stress, a Critical Vicious Circle in Neurodegenerative Tauopathies?Valuing the Endangered Species Antirrhinum lopesianum: Neuroprotective Activities and Strategies for in vitro Plant PropagationMitoenergetic failure in Alzheimer disease.An integrated view of oxidative stress in aging: basic mechanisms, functional effects, and pathological considerations.Plasma methionine sulfoxide in persons with familial Alzheimer's disease mutations.Early diagnostics and therapeutics for Alzheimer's disease--how early can we get there?N-terminal cleavage of the mitochondrial fusion GTPase OPA1 occurs via a caspase-independent mechanism in cerebellar granule neurons exposed to oxidative or nitrosative stress.Cytoskeletal transport in the aging brain: focus on the cholinergic system.
P2860
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P2860
Is oxidative damage the fundamental pathogenic mechanism of Alzheimer's and other neurodegenerative diseases?
description
2002 nî lūn-bûn
@nan
2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
name
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@ast
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@en
type
label
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@ast
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@en
prefLabel
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@ast
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@en
P2093
P50
P1476
Is oxidative damage the fundam ...... er neurodegenerative diseases?
@en
P2093
Akihiko Nunomura
Atsushi Takeda
Craig S Atwood
Gjumrakch Aliev
Jesús Avila
Keisuke Hirai
Lawrence M Sayre
Rudolph J Castellani
P304
P356
10.1016/S0891-5849(02)01113-9
P577
2002-12-01T00:00:00Z