IGF-1 increases invasive potential of MCF 7 breast cancer cells and induces activation of latent TGF-β1 resulting in epithelial to mesenchymal transition.
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The role of endocytic Rab GTPases in regulation of growth factor signaling and the migration and invasion of tumor cellsEpithelial-to-mesenchymal transition in breast cancer: a role for insulin-like growth factor I and insulin-like growth factor-binding protein 3?Differential effect of growth factors on invasion and proliferation of endocrine resistant breast cancer cellsAntrodin C inhibits epithelial-to-mesenchymal transition and metastasis of breast cancer cells via suppression of Smad2/3 and β-catenin signaling pathwaysHyperglycemia enhances the proliferation of non-tumorigenic and malignant mammary epithelial cells through increased leptin/IGF1R signaling and activation of AKT/mTOR.Novel role of cannabinoid receptor 2 in inhibiting EGF/EGFR and IGF-I/IGF-IR pathways in breast cancer.Ubiquitin ligase Cbl-b represses IGF-I-induced epithelial mesenchymal transition via ZEB2 and microRNA-200c regulation in gastric cancer cellsDifferential subcellular and extracellular localisations of proteins required for insulin-like growth factor- and extracellular matrix-induced signalling events in breast cancer progressionAnalysis of the quantitative balance between insulin-like growth factor (IGF)-1 ligand, receptor, and binding protein levels to predict cell sensitivity and therapeutic efficacy.Insulin-like growth factor-I induces epithelial to mesenchymal transition via GSK-3β and ZEB2 in the BGC-823 gastric cancer cell lineSox2 suppresses the invasiveness of breast cancer cells via a mechanism that is dependent on Twist1 and the status of Sox2 transcription activity.Insulin/IGF-I signaling pathways enhances tumor cell invasion through bisecting GlcNAc N-glycans modulation. an interplay with E-cadherin.The role of the insulin-like growth factor-1 system in breast cancerFAK activation is required for IGF1R-mediated regulation of EMT, migration, and invasion in mesenchymal triple negative breast cancer cellsInsulin and IGF-1, but not 17β-estradiol, alter the subcellular localization of MIER1α in MCF7 breast carcinoma cells.Metformin: an emerging new therapeutic option for targeting cancer stem cells and metastasisCirculating primers enhance platelet function and induce resistance to antiplatelet therapy.Pregnancy associated plasma protein-A links pregnancy and melanoma progression by promoting cellular migration and invasionPlasmonic labeling of subcellular compartments in cancer cells: multiplexing with fine-tuned gold and silver nanoshells.Altered local and systemic immune profiles underlie lymph node metastasis in breast cancer patients.Complex temporal changes in TGFβ oncogenic signaling drive thyroid carcinogenesis in a mouse modelIGF-IR cooperates with ERα to inhibit breast cancer cell aggressiveness by regulating the expression and localisation of ECM moleculesEpithelial-to-mesenchymal transition induced by TGF-β1 is mediated by AP1-dependent EpCAM expression in MCF-7 cells.RNA-Seq and Network Analysis Revealed Interacting Pathways in TGF-β-Treated Lung Cancer Cell Lines.Transforming growth factor β1 signal is crucial for dedifferentiation of cancer cells to cancer stem cells in osteosarcoma.Analysis of the MMP-dependent and independent functions of tissue inhibitor of metalloproteinase-2 on the invasiveness of breast cancer cells.Transforming growth factor-β, matrix metalloproteinases, and urokinase-type plasminogen activator interaction in the cancer epithelial to mesenchymal transition.Anti-TNFα alters the natural history of experimental Crohn's disease in rats when begun early, but not late, in disease.Core-shell hydrogel beads with extracellular matrix for tumor spheroid formation.Negative feedback loop between ZBTB7A and TGF-β in breast cancer.Phenformin inhibits growth and epithelial-mesenchymal transition of ErbB2-overexpressing breast cancer cells through targeting the IGF1R pathwayInsulin-like growth factor-1 activates different catalytic subunits p110 of PI3K in a cell-type-dependent manner to induce lipogenesis-dependent epithelial-mesenchymal transition through the regulation of ADAM10 and ADAM17.Genes involved in the immediate early response and epithelial-mesenchymal transition are regulated by adipocytokines in the female reproductive tract.Metformin inhibits the proliferation and metastasis of osteosarcoma cells by suppressing the phosphorylation of Akt.Molecular Signatures of the Insulin-like Growth Factor 1-mediated Epithelial-Mesenchymal Transition in Breast, Lung and Gastric Cancers
P2860
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P2860
IGF-1 increases invasive potential of MCF 7 breast cancer cells and induces activation of latent TGF-β1 resulting in epithelial to mesenchymal transition.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@ast
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@en
type
label
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@ast
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@en
prefLabel
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@ast
IGF-1 increases invasive poten ...... ial to mesenchymal transition.
@en
P2860
P356
P1476
IGF-1 increases invasive poten ...... lial to mesenchymal transition
@en
P2093
Logan A Walsh
Sashko Damjanovski
P2860
P2888
P356
10.1186/1478-811X-9-10
P577
2011-05-02T00:00:00Z
P6179
1001672743