A higher oxidative status accelerates senescence and aggravates age-dependent disorders in SAMP strains of mice.
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An overview of underlying causes and animal models for the study of age-related degenerative disorders of the spine and synovial jointsStem cell treatment for Alzheimer's diseaseDisruptive chemicals, senescence and immortalityOverexpression of X-Linked Inhibitor of Apoptotic Protein (XIAP) reduces age-related neuronal degeneration in the mouse cochleaProteomic analysis for protein carbonyl as an indicator of oxidative damage in senescence-accelerated mice.Effect of antioxidant-enriched diets on glutathione redox status in tissue homogenates and mitochondria of the senescence-accelerated mouseComparison of thiol redox state of mitochondria and homogenates of various tissues between two strains of mice with different longevities.Habitual exercise plus dietary supplementation with milk fat globule membrane improves muscle function deficits via neuromuscular development in senescence-accelerated miceTea catechin ingestion combined with habitual exercise suppresses the aging-associated decline in physical performance in senescence-accelerated mice.Melatonin in mitochondrial dysfunction and related disordersMelatonin protects lung mitochondria from agingSenescence of chondrocytes in aging articular cartilage: GADD45β mediates p21 expression in association with C/EBPβ in senescence-accelerated mice.Aging decreases antioxidant effects and increases lipid peroxidation in the Apolipoprotein E deficient mouseThe role of mitochondria in brain aging and the effects of melatonin.Mitochondrial metabolic failure in telomere attrition-provoked aging of bone marrow mesenchymal stem cells.The senescence-accelerated mouse prone-8 (SAM-P8) oxidative stress is associated with upregulation of renal NADPH oxidase system.Neuropathology and neurochemistry of nonmotor symptoms in Parkinson's disease.A calcineurin antagonist-free induction/maintenance strategy for immunosuppression in elderly recipients of renal allografts from elderly cadaver donors: long-term results from a prospective single centre trial.Molecular characterization of an allelic series of mutations in the mouse Nox3 geneAntioxidant activity of oligosaccharide ester extracted from Polygala tenuifolia roots in senescence-accelerated mice.Low in vivo brain glucose consumption and high oxidative stress in accelerated aging.Chronic administration of melatonin reduces cerebral injury biomarkers in SAMP8.Sildenafil Decreases BACE1 and Cathepsin B Levels and Reduces APP Amyloidogenic Processing in the SAMP8 Mouse.Aging-associated changes in physical performance and energy metabolism in the senescence-accelerated mouse.Chronic melatonin treatment reduces the age-dependent inflammatory process in senescence-accelerated mice.Longitudinal Performance of Senescence Accelerated Mouse Prone-Strain 8 (SAMP8) Mice in an Olfactory-Visual Water Maze Challenge
P2860
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P2860
A higher oxidative status accelerates senescence and aggravates age-dependent disorders in SAMP strains of mice.
description
2002 nî lūn-bûn
@nan
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
A higher oxidative status acce ...... rders in SAMP strains of mice.
@ast
A higher oxidative status acce ...... rders in SAMP strains of mice.
@en
type
label
A higher oxidative status acce ...... rders in SAMP strains of mice.
@ast
A higher oxidative status acce ...... rders in SAMP strains of mice.
@en
prefLabel
A higher oxidative status acce ...... rders in SAMP strains of mice.
@ast
A higher oxidative status acce ...... rders in SAMP strains of mice.
@en
P1476
A higher oxidative status acce ...... rders in SAMP strains of mice.
@en
P2093
Masanori Hosokawa
P304
P356
10.1016/S0047-6374(02)00091-X
P577
2002-11-01T00:00:00Z