Mechanism of neuromuscular dysfunction in Krabbe disease. - Wikidata - wikimedia - TriplyDB

Mechanism of neuromuscular dysfunction in Krabbe disease.

Mechanism of neuromuscular dysfunction in Krabbe disease.

http://www.wikidata.org/entity/Q35020269

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Lysosomal Re-acidification Prevents Lysosphingolipid-Induced Lysosomal Impairment and Cellular Toxicity How membrane dysfunction influences neuronal survival pathways in sphingolipid storage disorders Clinical, electrophysiological, and biochemical markers of peripheral and central nervous system disease in canine globoid cell leukodystrophy (Krabbe's disease)Synaptic failure: The achilles tendon of sphingolipidoses Ultrastructural Characterization of the Lower Motor System in a Mouse Model of Krabbe Disease.Lysosphingolipids and sphingolipidoses: Psychosine in Krabbe's disease.Biochemical, cell biological, pathological, and therapeutic aspects of Krabbe's disease.Axonal pathology in Krabbe's disease: The cytoskeleton as an emerging therapeutic target.Substrate reduction therapy for Krabbe's disease.Perspective on innovative therapies for globoid cell leukodystrophy.Diffusion tensor imaging analysis of the brain in the canine model of Krabbe disease.Analysis of age-related changes in psychosine metabolism in the human brain.α-Synuclein interacts directly but reversibly with psychosine: implications for α-synucleinopathies

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Mechanism of neuromuscular dysfunction in Krabbe disease.

http://www.wikidata.org/entity/Q35020269

description

2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Mechanism of neuromuscular dysfunction in Krabbe disease.

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Mechanism of neuromuscular dysfunction in Krabbe disease.

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Mechanism of neuromuscular dysfunction in Krabbe disease.

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Mechanism of neuromuscular dysfunction in Krabbe disease.

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Mechanism of neuromuscular dysfunction in Krabbe disease.

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Mechanism of neuromuscular dysfunction in Krabbe disease.

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JNEUROSCI.2431-14.2015

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Journal of Neuroscience

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Mechanism of neuromuscular dysfunction in Krabbe disease

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Aurora Lopez-Rosas

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Charles Vite

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Erick Maravilla

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Ernesto R Bongarzone

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James Jeffries

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Jesus Garcia

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John Monge

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Tammy Tamayo

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Tuba Sural-Fehr

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Mechanism of activation of protein kinase B by insulin and IGF-1

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Krabbe disease: genetic aspects and progress toward therapy

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

Signalling pathways that mediate skeletal muscle hypertrophy and atrophy

Transplantation of umbilical-cord blood in babies with infantile Krabbe's disease

A protein kinase B-dependent and rapamycin-sensitive pathway controls skeletal muscle growth but not fiber type specification.

Molecular cloning and expression of cDNA for murine galactocerebrosidase and mutation analysis of the twitcher mouse, a model of Krabbe's disease

Imaging neuronal subsets in transgenic mice expressing multiple spectral variants of GFP

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

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1606-1616

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scholarly article

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10.1523/JNEUROSCI.2431-14.2015

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Richard van Breemen

Ludovic D'Auria

Michael S Marshall

Ludovico Cantuti

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2015-01-01T00:00:00Z

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25632136

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4308604

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