Valproic acid treatment inhibits hypoxia-inducible factor 1α accumulation and protects against burn-induced gut barrier dysfunction in a rodent model
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Creating a "Prosurvival Phenotype" Through Histone Deacetylase Inhibition: Past, Present, and FuturemicroRNA-98 mediated microvascular hyperpermeability during burn shock phase via inhibiting FIH-1.Butyrate reduces high-fat diet-induced metabolic alterations, hepatic steatosis and pancreatic beta cell and intestinal barrier dysfunctions in prediabetic mice.Valproic acid inhibits the angiogenic potential of cervical cancer cells via HIF-1α/VEGF signals.Remote Burn Injury Increases Pulmonary Histone Deacetylase 1 and Reduces Histone Acetylation.Alcohol Modulation of the Postburn Hepatic Response.
P2860
Valproic acid treatment inhibits hypoxia-inducible factor 1α accumulation and protects against burn-induced gut barrier dysfunction in a rodent model
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2013 nî lūn-bûn
@nan
2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
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Valproic acid treatment inhibi ...... dysfunction in a rodent model
@ast
Valproic acid treatment inhibi ...... dysfunction in a rodent model
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type
label
Valproic acid treatment inhibi ...... dysfunction in a rodent model
@ast
Valproic acid treatment inhibi ...... dysfunction in a rodent model
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Valproic acid treatment inhibi ...... dysfunction in a rodent model
@ast
Valproic acid treatment inhibi ...... dysfunction in a rodent model
@en
P2093
P2860
P1433
P1476
Valproic acid treatment inhibi ...... dysfunction in a rodent model
@en
P2093
Hong-Min Luo
Jiang-Yang Lu
Ming-Hua Du
P2860
P304
P356
10.1371/JOURNAL.PONE.0077523
P407
P577
2013-10-17T00:00:00Z