What cardiovascular defect does my prenatal mouse mutant have, and why?
about
SWI/SNF complexes containing Brahma or Brahma-related gene 1 play distinct roles in smooth muscle development.Gene trap disruption of the mouse heparan sulfate 6-O-endosulfatase gene, Sulf2Line-scanning particle image velocimetry: an optical approach for quantifying a wide range of blood flow speeds in live animalsENU-induced mutation in the DNA-binding domain of KLF3 reveals important roles for KLF3 in cardiovascular development and function in miceTrkb signaling in pericytes is required for cardiac microvessel stabilizationGene targeting reveals a widespread role for the high-mobility-group transcription factor Sox11 in tissue remodelingDefects in yolk sac vasculogenesis, chorioallantoic fusion, and embryonic axis elongation in mice with targeted disruption of Yap65Embryonic arrest at midgestation and disruption of Notch signaling produced by the absence of both epsin 1 and epsin 2 in miceOtt1 (Rbm15) is essential for placental vascular branching morphogenesis and embryonic development of the heart and spleenThe trafficking protein Tmed2/p24beta(1) is required for morphogenesis of the mouse embryo and placentaHigh resolution ultrasound-guided microinjection for interventional studies of early embryonic and placental development in vivo in mice.Cell-autonomous requirement for beta1 integrin in endothelial cell adhesion, migration and survival during angiogenesis in mice.Piezo1, a mechanically activated ion channel, is required for vascular development in mice.Vascular remodeling of the mouse yolk sac requires hemodynamic force.Algorithms for improved 3-D reconstruction of live mammalian embryo vasculature from optical coherence tomography data.Identification of the heart as the critical site of adenosine mediated embryo protection.A membrane associated mCherry fluorescent reporter line for studying vascular remodeling and cardiac function during murine embryonic development.Trigenic neural crest-restricted Smad7 over-expression results in congenital craniofacial and cardiovascular defects.Deletion of exon 20 of the Familial Dysautonomia gene Ikbkap in mice causes developmental delay, cardiovascular defects, and early embryonic lethalitySox7 is regulated by ETV2 during cardiovascular developmentDosage-dependent effects of Akt1/protein kinase Balpha (PKBalpha) and Akt3/PKBgamma on thymus, skin, and cardiovascular and nervous system development in mice.A hypomorphic lsd1 allele results in heart development defects in miceVascular endothelial growth factor receptor-2 promotes the development of the lymphatic vasculature.Bioenergetics, mitochondria, and cardiac myocyte differentiationCardiovascular development and survival during gestation in the Ts65Dn mouse model for Down syndrome.Adverse and protective influences of adenosine on the newborn and embryo: implications for preterm white matter injury and embryo protectionMethionine synthase reductase deficiency results in adverse reproductive outcomes and congenital heart defects in mice.Prdm6 is essential for cardiovascular development in vivoMice carrying a hypomorphic Evi1 allele are embryonic viable but exhibit severe congenital heart defectsThe ubiquitin ligase ASB4 promotes trophoblast differentiation through the degradation of ID2Redundant and dosage sensitive requirements for Fgf3 and Fgf10 in cardiovascular developmentPax3 is essential for normal cardiac neural crest morphogenesis but is not required during migration nor outflow tract septation.Persistent Noggin arrests cardiomyocyte morphogenesis and results in early in utero lethality.The permeability transition pore controls cardiac mitochondrial maturation and myocyte differentiation.Cardiovascular expression of the mouse WNK1 gene during development and adulthood revealed by a BAC reporter assay.Differential role of PKA catalytic subunits in mediating phenotypes caused by knockout of the Carney complex gene Prkar1a.Maternal and zygotic Zfp57 modulate NOTCH signaling in cardiac development.Casz1 is required for cardiomyocyte G1-to-S phase progression during mammalian cardiac developmentLack of endothelial cell survivin causes embryonic defects in angiogenesis, cardiogenesis, and neural tube closure.Regulation of cardiovascular development by adenosine and adenosine-mediated embryo protection.
P2860
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P2860
What cardiovascular defect does my prenatal mouse mutant have, and why?
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
What cardiovascular defect does my prenatal mouse mutant have, and why?
@ast
What cardiovascular defect does my prenatal mouse mutant have, and why?
@en
type
label
What cardiovascular defect does my prenatal mouse mutant have, and why?
@ast
What cardiovascular defect does my prenatal mouse mutant have, and why?
@en
prefLabel
What cardiovascular defect does my prenatal mouse mutant have, and why?
@ast
What cardiovascular defect does my prenatal mouse mutant have, and why?
@en
P2093
P2860
P356
P1433
P1476
What cardiovascular defect does my prenatal mouse mutant have, and why?
@en
P2093
Agnieszka Kruzynska-Frejtag
Michal Machnicki
Paige L Kneer
Simon J Conway
Srinagesh V Koushik
P2860
P356
10.1002/GENE.10152
P577
2003-01-01T00:00:00Z