The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis. - Wikidata - wikimedia - TriplyDB

The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

http://www.wikidata.org/entity/Q35025300

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Mutation in CPT1C Associated With Pure Autosomal Dominant Spastic Paraplegia Brain Lipotoxicity of Phytanic Acid and Very Long-chain Fatty Acids. Harmful Cellular/Mitochondrial Activities in Refsum Disease and X-Linked Adrenoleukodystrophy Hypothalamic malonyl-CoA and the control of food intake Structural characterization of the regulatory domain of brain carnitine palmitoyltransferase 1 Reduction of brain mitochondrial β-oxidation impairs complex I and V in chronic alcohol intake: the underlying mechanism for neurodegeneration Increasing fatty acid oxidation remodels the hypothalamic neurometabolome to mitigate stress and inflammation CPT1c is localized in endoplasmic reticulum of neurons and has carnitine palmitoyltransferase activity Acyl coenzyme A thioesterase 7 regulates neuronal fatty acid metabolism to prevent neurotoxicity.High grade glioblastoma is associated with aberrant expression of ZFP57, a protein involved in gene imprinting, and of CPT1A and CPT1C that regulate fatty acid metabolism The CPT1C 5'UTR contains a repressing upstream open reading frame that is regulated by cellular energy availability and AMPK A ketone ester diet increases brain malonyl-CoA and Uncoupling proteins 4 and 5 while decreasing food intake in the normal Wistar Rat.Hypothalamic AMPK and fatty acid metabolism mediate thyroid regulation of energy balance.Carnitine palmitoyltransferase 1A (CPT1A): a transcriptional target of PAX3-FKHR and mediates PAX3-FKHR-dependent motility in alveolar rhabdomyosarcoma cells Carnitine Palmitoyltransferase 1b Deficiency Protects Mice from Diet-Induced Insulin Resistance Metabolomic profiling reveals a role for CPT1c in neuronal oxidative metabolism Investigation of genes important in neurodevelopment disorders in adult human brain.Dietary fat content and fiber type modulate hind gut microbial community and metabolic markers in the pig.Leptin activates hypothalamic acetyl-CoA carboxylase to inhibit food intake.Glial β-oxidation regulates Drosophila energy metabolism.Carnitine palmitoyltransferase 1C promotes cell survival and tumor growth under conditions of metabolic stress.Important roles of brain-specific carnitine palmitoyltransferase and ceramide metabolism in leptin hypothalamic control of feeding Pregnancy induces resistance to the anorectic effect of hypothalamic malonyl-CoA and the thermogenic effect of hypothalamic AMPK inhibition in female rats.Hypothalamic malonyl-CoA triggers mitochondrial biogenesis and oxidative gene expression in skeletal muscle: Role of PGC-1alpha.Differentially expressed genes in adipocytokine signaling pathway of adipose tissue in pregnancy.The Origin and Diversity of Cpt1 Genes in Vertebrate Species.Targeted chemical-genetic regulation of protein stability in vivo Ceramide levels regulated by carnitine palmitoyltransferase 1C control dendritic spine maturation and cognition.Genome-wide association studies for feed intake and efficiency in two laying periods of chickens.Regulation of hypothalamic malonyl-CoA by central glucose and leptin.The metabolome regulates the epigenetic landscape during naive-to-primed human embryonic stem cell transition.Cisplatin-induced downregulation of OCTN2 affects carnitine wasting Homozygous carnitine palmitoyltransferase 1b (muscle isoform) deficiency is lethal in the mouse Individuals with Metabolically Healthy Overweight/Obesity Have Higher Fat Utilization than Metabolically Unhealthy Individuals.Neurometabolic roles of ApoE and Ldl-R in mouse brain Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol.Malonyl-CoA mediates leptin hypothalamic control of feeding independent of inhibition of CPT-1a Molecular system bioenergetics: regulation of substrate supply in response to heart energy demands.Carnitine palmitoyltransferase 1A functions to repress FoxO transcription factors to allow cell cycle progression in ovarian cancer.Evidence for a role of LPGAT1 in influencing BMI and percent body fat in Native Americans.Sphingolipid metabolism and obesity-induced inflammation.

P2860

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P2860

The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

http://www.wikidata.org/entity/Q35025300

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2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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type

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The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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PNAS.0602205103

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Proceedings of the National Academy of Sciences of the United States of America

P1476

The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.

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P2093

Akira Suwa

http://www.w3.org/2001/XMLSchema#string

M Daniel Lane

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Makoto Asaumi

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Michael J Wolfgang

http://www.w3.org/2001/XMLSchema#string

Seung Hun Cha

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Shun-Ichiro Matsumoto

http://www.w3.org/2001/XMLSchema#string

Takeshi Kurama

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Teruhiko Shimokawa

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Yun Dai

http://www.w3.org/2001/XMLSchema#string

P2860

Acetyl-CoA carboxylase 2 mutant mice are protected against obesity and diabetes induced by high-fat/high-carbohydrate diets

Crystal structure of carnitine acetyltransferase and implications for the catalytic mechanism and fatty acid transport

A novel brain-expressed protein related to carnitine palmitoyltransferase I

Continuous fatty acid oxidation and reduced fat storage in mice lacking acetyl-CoA carboxylase 2

AMP-activated protein kinase plays a role in the control of food intake

AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamus

Inhibition of hypothalamic carnitine palmitoyltransferase-1 decreases food intake and glucose production

Disruption of neural signal transducer and activator of transcription 3 causes obesity, diabetes, infertility, and thermal dysregulation

Central nervous system control of food intake

Loss of stearoyl-CoA desaturase-1 function protects mice against adiposity

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7282-7287

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10.1073/PNAS.0602205103

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19

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103

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7129067

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16651524

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Malonyl-Coenzyme A

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1564279

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