Recent advances in the pathogenesis and drug action in periodic paralyses and related channelopathies.
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ChannelopathiesTherapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug DiscoverySplicing of the rSlo gene affects the molecular composition and drug response of Ca2+-activated K+ channels in skeletal muscleEmerging role of calcium-activated potassium channel in the regulation of cell viability following potassium ions challenge in HEK293 cells and pharmacological modulation.Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy.A 20-year-old Chinese man with recurrent hypokalemic periodic paralysis and delayed diagnosisPathophysiological role of omega pore current in channelopathiesThe impact of permanent muscle weakness on quality of life in periodic paralysis: a survey of 66 patientsTransient compartment-like syndrome and normokalaemic periodic paralysis due to a Ca(v)1.1 mutation.NaV1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery.Biophysics, pathophysiology, and pharmacology of ion channel gating pores.Commentary: A BK (Slo1) channel journey from molecule to physiologyMolecular cloning of ion channels in Felis catus that are related to periodic paralyses in man: a contribution to the understanding of the genetic susceptibility to feline neck ventroflexion and paralysis.The large-conductance calcium-activated potassium channel holds the key to the conundrum of familial hypokalemic periodic paralysis.Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents.Gender differences in penetrance and phenotype in hypokalemic periodic paralysis.Review of the Diagnosis and Treatment of Periodic Paralysis.Molecular structure and function of big calcium-activated potassium channels in skeletal muscle: pharmacological perspectives.Mechanisms Responsible for ω-Pore Currents in Cav Calcium Channel Voltage-Sensing Domains.Membrane dysfunction in Andersen-Tawil syndrome assessed by velocity recovery cycles.Protein kinase C theta (PKCθ) modulates the ClC-1 chloride channel activity and skeletal muscle phenotype: a biophysical and gene expression study in mouse models lacking the PKCθ.A New Cardiac Channelopathy: From Clinical Phenotypes to Molecular Mechanisms Associated With Na1.5 Gating PoresIncreased promoter activity as a mechanism in atypical normokalemic periodic paralysisA leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy
P2860
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P2860
Recent advances in the pathogenesis and drug action in periodic paralyses and related channelopathies.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Recent advances in the pathoge ...... s and related channelopathies.
@ast
Recent advances in the pathoge ...... s and related channelopathies.
@en
type
label
Recent advances in the pathoge ...... s and related channelopathies.
@ast
Recent advances in the pathoge ...... s and related channelopathies.
@en
prefLabel
Recent advances in the pathoge ...... s and related channelopathies.
@ast
Recent advances in the pathoge ...... s and related channelopathies.
@en
P2860
P356
P1476
Recent advances in the pathoge ...... s and related channelopathies.
@en
P2093
Diana Conte Camerino
Domenico Tricarico
P2860
P356
10.3389/FPHAR.2011.00008
P577
2011-02-28T00:00:00Z