Nitrosative stress elicited by nNOSµ delocalization inhibits muscle force in dystrophin-null mice
about
Nutraceuticals and Their Potential to Treat Duchenne Muscular Dystrophy: Separating the Credible from the ConjectureAnimal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapyTreatment with a nitric oxide-donating NSAID alleviates functional muscle ischemia in the mouse model of Duchenne muscular dystrophyAttempting to Compensate for Reduced Neuronal Nitric Oxide Synthase Protein with Nitrate Supplementation Cannot Overcome Metabolic Dysfunction but Rather Has Detrimental Effects in Dystrophin-Deficient mdx Muscle.The stress protein/chaperone Grp94 counteracts muscle disuse atrophy by stabilizing subsarcolemmal neuronal nitric oxide synthase.Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophyS-nitrosoglutathione reductase deficiency-induced S-nitrosylation results in neuromuscular dysfunction.iNOS ablation does not improve specific force of the extensor digitorum longus muscle in dystrophin-deficient mdx4cv mice.Deficient nitric oxide signalling impairs skeletal muscle growth and performance: involvement of mitochondrial dysregulation.Loss of nNOS inhibits compensatory muscle hypertrophy and exacerbates inflammation and eccentric contraction-induced damage in mdx mice.Duchenne muscular dystrophy gene therapy: Lost in translation?The FVB Background Does Not Dramatically Alter the Dystrophic Phenotype of Mdx MiceNitric oxide signalling pathway in Duchenne muscular dystrophy mice: up-regulation of L-arginine transporters.Duchenne muscular dystrophy gene therapy in the canine model.Novel mini-dystrophin gene dual adeno-associated virus vectors restore neuronal nitric oxide synthase expression at the sarcolemmaA simplified immune suppression scheme leads to persistent micro-dystrophin expression in Duchenne muscular dystrophy dogsDystrophin-compromised sarcoglycan-δ-knockout diaphragm requires full wild-type embryonic stem cell reconstitution for correction.Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Molecular dissection of dystrophin identifies the docking site for nNOSα2 and α3 helices of dystrophin R16 and R17 frame a microdomain in the α1 helix of dystrophin R17 for neuronal NOS binding.Truncated dystrophins reduce muscle stiffness in the extensor digitorum longus muscle of mdx mice.Exclusive skeletal muscle correction does not modulate dystrophic heart disease in the aged mdx model of Duchenne cardiomyopathyDystrophin restoration therapy improves both the reduced excitability and the force drop induced by lengthening contractions in dystrophic mdx skeletal muscle.Dual AAV therapy ameliorates exercise-induced muscle injury and functional ischemia in murine models of Duchenne muscular dystrophyS-nitrosation and ubiquitin-proteasome system interplay in neuromuscular disorders.Local inhibition of nitrergic activity in tenotomized rats accelerates muscle regeneration by increasing fiber area and decreasing central core lesions.Update on the treatment of Duchenne muscular dystrophy.Functional muscle ischemia in Duchenne and Becker muscular dystrophy.Nitric oxide synthase deficiency and the pathophysiology of muscular dystrophy.GSNOR Deficiency Enhances In Situ Skeletal Muscle Strength, Fatigue Resistance, and RyR1 S-Nitrosylation Without Impacting Mitochondrial Content and Activity.A Hypothesis for Examining Skeletal Muscle Biopsy-Derived Sarcolemmal nNOSμ as Surrogate for Enteric nNOSα Function.Nanotherapy for Duchenne muscular dystrophy.nNOS regulation of skeletal muscle fatigue and exercise performance.Uniform low-level dystrophin expression in the heart partially preserved cardiac function in an aged mouse model of Duchenne cardiomyopathy.A Five-Repeat Micro-Dystrophin Gene Ameliorated Dystrophic Phenotype in the Severe DBA/2J-mdx Model of Duchenne Muscular Dystrophy.Sodium nitrate alleviates functional muscle ischaemia in patients with Becker muscular dystrophy.Oxidative stress, NF-κB and the ubiquitin proteasomal pathway in the pathology of calpainopathy.Link between MHC Fiber Type and Restoration of Dystrophin Expression and Key Components of the DAPC by Tricyclo-DNA-Mediated Exon Skipping.Transient receptor potential channel 6 regulates abnormal cardiac S-nitrosylation in Duchenne muscular dystrophy.Micro-dystrophin gene therapy goes systemic in Duchenne muscular dystrophy patients.
P2860
Q28072269-EBD6C0FF-7161-40BE-8419-3102F67ABD12Q28084979-63FBBD47-4F64-403A-958D-0CC74D6F44EDQ28484851-877D74FC-4682-4405-978E-6D2BFEAE5451Q33586004-C4CDFFE0-3A80-4486-8C0B-DF468D8B3700Q33630112-81F181FA-01D9-4194-8435-D897B3DCDEEAQ33649312-686E05A6-BD14-4291-B504-E1C4DDE50AC3Q33862264-C78806D3-85BC-40F4-AA6F-CE2D8008672EQ33955159-B8C93B34-5015-45AD-9A13-6444EFFBEBF3Q34748781-DF5CA4A8-A509-40CE-9FC1-D4C9EF0AA52BQ34764061-9186D9D3-C23E-4AFF-AE3A-734002B23A72Q35052506-9B0B5E59-B8C3-4962-AD27-4D1AD6580D34Q35117470-87B9B209-B827-4328-928F-B11A22C3E8DDQ35195095-792409C7-97FB-469D-9232-5C9D3435E576Q35641673-C3C6BC66-A8D7-45F9-9B61-54109EE28337Q35678425-D8BF1E19-15E4-40AB-A049-0903F52F7319Q35751578-E1D63B1C-CE06-4340-A6FC-166E212E300EQ35941963-F1208F43-49C5-4C3F-8741-8A92328F33E2Q36422465-45AC0442-21DD-445F-A119-51FE51072533Q36535281-F49A37CA-2596-4C25-8D9E-8775DBC52C72Q36535303-F8EA3B06-7596-4629-B902-6B46E5D56F66Q36600235-0D2CD311-A595-4972-947B-FE48B5AC6542Q36908212-CAEE8642-1773-45E6-A03E-3BFE2A2FC2C3Q37111473-4E902871-1ACE-445B-814E-E42C36FECFAFQ37111962-30BCEF9B-7910-4C2D-9BAB-3C64E20A6631Q37592652-6D8C4A5A-544B-4E3A-BF75-F2899CF01087Q37677198-F9967F40-48D2-42B4-B818-83A4E41F5980Q38074834-D34571C3-A11B-4E38-8335-C233BED2C2E1Q38175976-F07B1FEB-4387-4CB3-980D-AE4934E7EF82Q38246459-49ED29B6-B30A-481E-8579-9DCF0897AB62Q38412593-EF993487-4C94-4FA3-95D8-9040AC008F97Q38973194-FADFCFEB-02CE-4679-AB13-4C2A62EC1C4EQ39233936-59EBCB89-4C20-445B-856B-D4252A777334Q39310461-F22FC718-C7A1-46C9-BA37-175CA8EE94A2Q40445952-F9EC4A3A-06AF-4DFF-B3C1-611FCC7C63CFQ41171772-A5C04020-8C69-4E72-A5A8-DD897186B4B2Q46088505-598433DD-A11C-44B6-90CA-D76031F05C40Q46262861-C83580BB-FF2D-4084-ACAC-9DBB5CE9DC87Q47288511-157E23FD-67B3-4F80-972E-6950CAEC0F6BQ47350159-B8710F94-769E-4875-BE4F-FAA6E7D64807Q49797651-812FAD41-0B0C-4D07-B7C5-CBCE230D3AF2
P2860
Nitrosative stress elicited by nNOSµ delocalization inhibits muscle force in dystrophin-null mice
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Nitrosative stress elicited by ...... force in dystrophin-null mice
@ast
Nitrosative stress elicited by ...... force in dystrophin-null mice
@en
type
label
Nitrosative stress elicited by ...... force in dystrophin-null mice
@ast
Nitrosative stress elicited by ...... force in dystrophin-null mice
@en
prefLabel
Nitrosative stress elicited by ...... force in dystrophin-null mice
@ast
Nitrosative stress elicited by ...... force in dystrophin-null mice
@en
P2093
P2860
P356
P1476
Nitrosative stress elicited by ...... force in dystrophin-null mice
@en
P2093
Chady H Hakim
Yongping Yue
P2860
P356
10.1002/PATH.2799
P577
2010-10-28T00:00:00Z