The francisella intracellular life cycle: toward molecular mechanisms of intracellular survival and proliferation.
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Structure-Function Analysis of DipA, a Francisella tularensis Virulence Factor Required for Intracellular ReplicationEvidence Suggesting That Francisella tularensis O-Antigen Capsule Contains a Lipid A-Like Molecule That Is Structurally Distinct from the More Abundant Free Lipid AEstablishment of three Francisella infections in zebrafish embryos at different temperatures.Francisella requires dynamic type VI secretion system and ClpB to deliver effectors for phagosomal escape.Isolation of F. novicida-Containing Phagosome from Infected Human Monocyte Derived Macrophages.DotU and VgrG, core components of type VI secretion systems, are essential for Francisella LVS pathogenicityCoordinate control of virulence gene expression in Francisella tularensis involves direct interaction between key regulatorsDifferential expression of microRNAs in Francisella tularensis-infected human macrophages: miR-155-dependent downregulation of MyD88 inhibits the inflammatory response.Identification of disulfide bond isomerase substrates reveals bacterial virulence factors.The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infectionImportance of branched-chain amino acid utilization in Francisella intracellular adaptationFrancisella-arthropod vector interaction and its role in patho-adaptation to infect mammalsIntra-Vacuolar Proliferation of F. Novicida within H. VermiformisAtomic structure of T6SS reveals interlaced array essential to functionIglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVSUbiquitous promoter-localization of essential virulence regulators in Francisella tularensisFrancisella tularensis inhibits the intrinsic and extrinsic pathways to delay constitutive apoptosis and prolong human neutrophil lifespan.Using host-pathogen protein interactions to identify and characterize Francisella tularensis virulence factors.FmvB: A Francisella tularensis Magnesium-Responsive Outer Membrane Protein that Plays a Role in Virulence.Natural IgM mediates complement-dependent uptake of Francisella tularensis by human neutrophils via complement receptors 1 and 3 in nonimmune serum.Link between intraphagosomal biotin and rapid phagosomal escape in FrancisellaTemporal transcriptional response during infection of type II alveolar epithelial cells with Francisella tularensis live vaccine strain (LVS) supports a general host suppression and bacterial uptake by macropinocytosis.Infection with Francisella tularensis LVS clpB leads to an altered yet protective immune responseAutophagic clearance of bacterial pathogens: molecular recognition of intracellular microorganismsNeutrophils: potential therapeutic targets in tularemia?Quantitative assessment of cytosolic Salmonella in epithelial cells.Lack of OxyR and KatG Results in Extreme Susceptibility of Francisella tularensis LVS to Oxidative Stress and Marked Attenuation In vivo.A new dye uptake assay to test the activity of antibiotics against intracellular Francisella tularensis.Proteins involved in Francisella tularensis survival and replication inside macrophages.At the crossroads: communication of bacteria-containing vacuoles with host organelles.Comparative proteome profiling of host-pathogen interactions: insights into the adaptation mechanisms of Francisella tularensis in the host cell environment.Asparagine assimilation is critical for intracellular replication and dissemination of Francisella.Increased Resistance to Intradermal Francisella tularensis LVS Infection by Inactivation of the Sts Phosphatases.A mutagenesis-based approach identifies amino acids in the N-terminal part of Francisella tularensis IglE that critically control Type VI system-mediated secretion.Exploiting a natural auxotrophy for genetic selection.The contribution of the glycine cleavage system to the pathogenesis of Francisella tularensis.Special Topic on Francisella tularensis and TularemiaA case of tularemia after an endurance run in a non-endemic region.Deletion of the Major Facilitator Superfamily Transporter fptB Alters Host Cell Interactions and Attenuates Virulence of Type A Francisella tularensis.The orange spotted cockroach (Blaptica dubia, Serville 1839) is a permissive experimental host for Francisella tularensis.
P2860
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P2860
The francisella intracellular life cycle: toward molecular mechanisms of intracellular survival and proliferation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The francisella intracellular ...... ar survival and proliferation.
@ast
The francisella intracellular ...... ar survival and proliferation.
@en
type
label
The francisella intracellular ...... ar survival and proliferation.
@ast
The francisella intracellular ...... ar survival and proliferation.
@en
prefLabel
The francisella intracellular ...... ar survival and proliferation.
@ast
The francisella intracellular ...... ar survival and proliferation.
@en
P2860
P356
P1476
The francisella intracellular ...... ar survival and proliferation.
@en
P2093
Audrey Chong
Jean Celli
P2860
P356
10.3389/FMICB.2010.00138
P577
2010-12-28T00:00:00Z