Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis - Wikidata - wikimedia - TriplyDB

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

http://www.wikidata.org/entity/Q35032869

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Protective links between vitamin D, inflammatory bowel disease and colon cancer Breast Milk and Solid Food Shaping Intestinal Immunity Vitamin D as an Immunomodulator: Risks with Deficiencies and Benefits of Supplementation Intestinal Permeability in Inflammatory Bowel Disease: Pathogenesis, Clinical Evaluation, and Therapy of Leaky Gut Vitamin D and inflammatory bowel disease Vitamin D differentially regulates Salmonella-induced intestine epithelial autophagy and interleukin-1β expression Role of Vitamin D in the Hygiene Hypothesis: The Interplay between Vitamin D, Vitamin D Receptors, Gut Microbiota, and Immune Response Lactobacillus casei Zhang and vitamin K2 prevent intestinal tumorigenesis in mice via adiponectin-elevated different signaling pathways.Vitamin D/VDR, Probiotics, and Gastrointestinal Diseases Vitamin D: Metabolism, Molecular Mechanism of Action, and Pleiotropic Effects.Exploring gut microbes in human health and disease: Pushing the envelope.Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model Vitamin D receptor pathway is required for probiotic protection in colitis.The role of vitamin D in reducing gastrointestinal disease risk and assessment of individual dietary intake needs: Focus on genetic and genomic technologies.Role of Autophagy in the Maintenance of Intestinal Homeostasis.Vitamin D-induced up-regulation of human keratinocyte cathelicidin anti-microbial peptide expression involves retinoid X receptor α.Genotype-Phenotype Associations of the CD-Associated Single Nucleotide Polymorphism within the Gene Locus Encoding Protein Tyrosine Phosphatase Non-Receptor Type 22 in Patients of the Swiss IBD Cohort Tight junction CLDN2 gene is a direct target of the vitamin D receptor.The role of 1,25-dyhydroxyvitamin D3 in mouse liver ischemia reperfusion injury: regulation of autophagy through activation of MEK/ERK signaling and PTEN/PI3K/Akt/mTORC1 signaling.Characterization of candidate genes in inflammatory bowel disease-associated risk loci.Intestinal Autophagy and Its Pharmacological Control in Inflammatory Bowel Disease.Autophagy and checkpoints for intracellular pathogen defense.The effect of polymeric formula on enterocyte differentiation.Human cytomegalovirus infection downregulates vitamin-D receptor in mammalian cells.Vitamin D Receptor-Dependent Signaling Protects Mice From Dextran Sulfate Sodium-Induced Colitis.Flow cytometry detection of vitamin D receptor changes during vitamin D treatment in Crohn's disease.1,25(OH)2 D3 attenuates hepatic steatosis by inducing autophagy in mice.Salmonella enteritidis Effector AvrA Stabilizes Intestinal Tight Junctions via the JNK Pathway.Genetic control of autophagy underlies pathogenesis of inflammatory bowel disease.Role of autophagy in the pathogenesis of inflammatory bowel disease Interplay of host genetics and gut microbiota underlying the onset and clinical presentation of inflammatory bowel disease.Vitamin D receptor is a novel transcriptional regulator for Axin1.The Role of Vitamin D and Vitamin D Receptors in Colon Cancer.A vitamin D receptor agonist converts CD4+ T cells to Foxp3+ regulatory T cells in patients with ulcerative colitis.Time for epithelial sensing of vitamin D to step into the limelight.Vitamin D3 induces vitamin D receptor and HDAC11 binding to relieve the promoter of the tight junction proteins.Gut inflammation and dysbiosis in human motor neuron disease.Inflammatory Bowel Disease: Influence and Implications in Reproduction.Effect of Long Noncoding RNA H19 Overexpression on Intestinal Barrier Function and Its Potential Role in the Pathogenesis of Ulcerative Colitis.Autophagy Genes of Host Responds to Disruption of Gut Microbial Community by Antibiotics.

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P2860

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

http://www.wikidata.org/entity/Q35032869

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@ast

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@en

type

label

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@ast

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@en

prefLabel

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@ast

Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

@en

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Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis

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David Zhou

http://www.w3.org/2001/XMLSchema#string

Di Chen

http://www.w3.org/2001/XMLSchema#string

Elaine O Petrof

http://www.w3.org/2001/XMLSchema#string

Erika C Claud

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Eugene B Chang

http://www.w3.org/2001/XMLSchema#string

Geert Carmeliet

http://www.w3.org/2001/XMLSchema#string

Rong Lu

http://www.w3.org/2001/XMLSchema#string

Shaoping Wu

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Yinglin Xia

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Yong-Guo Zhang

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P2860

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

Resident enteric bacteria are necessary for development of spontaneous colitis and immune system activation in interleukin-10-deficient mice

Duodenal calcium absorption in vitamin D receptor-knockout mice: functional and molecular aspects

Autophagy in immunity and inflammation

Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface

Vitamin D and the epidemiology of upper gastrointestinal cancers: a critical analysis of the current evidence

Multi-scale in vivo systems analysis reveals the influence of immune cells on TNF-α-induced apoptosis in the intestinal epithelium

Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3

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1082-1094

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