Defective viral genomes arising in vivo provide critical danger signals for the triggering of lung antiviral immunity - Wikidata - wikimedia - TriplyDB

Defective viral genomes arising in vivo provide critical danger signals for the triggering of lung antiviral immunity

Defective viral genomes arising in vivo provide critical danger signals for the triggering of lung antiviral immunity

http://www.wikidata.org/entity/Q35034212

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Cloned Defective Interfering Influenza RNA and a Possible Pan-Specific Treatment of Respiratory Virus Diseases Defective viral genomes: critical danger signals of viral infections Evidence for a Novel Mechanism of Influenza Virus-Induced Type I Interferon Expression by a Defective RNA-Encoded Protein Influenza virus activation of the interferon system lncRHOXF1, a Long Noncoding RNA from the X Chromosome That Suppresses Viral Response Genes during Development of the Early Human Placenta Persistent RNA virus infections: do PAMPS drive chronic disease?Collective Infectious Units in Viruses Parallel ClickSeq and Nanopore sequencing elucidates the rapid evolution of defective-interfering RNAs in Flock House virus.Biological activities of 'noninfectious' influenza A virus particles Flaviviral RNAs: weapons and targets in the war between virus and host.Immunostimulatory Defective Viral Genomes from Respiratory Syncytial Virus Promote a Strong Innate Antiviral Response during Infection in Mice and Humans.IFN-β-inducing, unusual viral RNA species produced by paramyxovirus infection accumulated into distinct cytoplasmic structures in an RNA-type-dependent manner In Vivo RNAi Screening Identifies MDA5 as a Significant Contributor to the Cellular Defense against Influenza A Virus Activity of Uncleaved Caspase-8 Controls Anti-bacterial Immune Defense and TLR-Induced Cytokine Production Independent of Cell Death Identification of a Natural Viral RNA Motif That Optimizes Sensing of Viral RNA by RIG-I.Late regulation of immune genes and microRNAs in circulating leukocytes in a pig model of influenza A (H1N2) infection.Propagation and Characterization of Influenza Virus Stocks That Lack High Levels of Defective Viral Genomes and Hemagglutinin Mutations Plasmacytoid Dendritic Cells Require Direct Infection To Sustain the Pulmonary Influenza A Virus-Specific CD8 T Cell Response.Low-Fidelity Polymerases of Alphaviruses Recombine at Higher Rates To Overproduce Defective Interfering Particles.Spatial-Temporal Patterns of Viral Amplification and Interference Initiated by a Single Infected Cell.Deficiency of melanoma differentiation-associated protein 5 results in exacerbated chronic postviral lung inflammation.Oncolytic Immunotherapy: Dying the Right Way is a Key to Eliciting Potent Antitumor Immunity.Defective interfering viruses and their impact on vaccines and viral vectors.Newcastle Disease Virus Establishes Persistent Infection in Tumor Cells In Vitro: Contribution of the Cleavage Site of Fusion Protein and Second Sialic Acid Binding Site of Hemagglutinin-Neuraminidase.Single-cell studies of IFN-β promoter activation by wild-type and NS1-defective influenza A viruses.Attenuation of RNA viruses by redirecting their evolution in sequence space.Human IFIT1 Inhibits mRNA Translation of Rubulaviruses but Not Other Members of the Paramyxoviridae Family.Activation of the interferon induction cascade by influenza a viruses requires viral RNA synthesis and nuclear export.An unbiased genetic screen reveals the polygenic nature of the influenza virus anti-interferon response.Targeting Pattern Recognition Receptors (PRR) for Vaccine Adjuvantation: From Synthetic PRR Agonists to the Potential of Defective Interfering Particles of Viruses.Population Diversity and Collective Interactions during Influenza Virus Infection.Revisiting the concept of a cytopathic viral infection.Loss of Sendai virus C protein leads to accumulation of RIG-I immunostimulatory defective interfering RNA.The innate immune response to RSV: Advances in our understanding of critical viral and host factors.Reduced accumulation of defective viral genomes contributes to severe outcome in influenza virus infected patients.Replication defective viral genomes exploit a cellular pro-survival mechanism to establish paramyxovirus persistence.A single amino acid substitution within the Paramyxovirus Sendai virus nucleoprotein is a critical determinant for production of IFN-beta-inducing copyback-type defective interfering genomes.Active Ebola Virus Replication and Heterogeneous Evolutionary Rates in EVD Survivors.Extreme heterogeneity of influenza virus infection in single cells.Low-fidelity Venezuelan equine encephalitis virus polymerase mutants to improve live-attenuated vaccine safety and efficacy.

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Defective viral genomes arising in vivo provide critical danger signals for the triggering of lung antiviral immunity

http://www.wikidata.org/entity/Q35034212

description

2013 nî lūn-bûn

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2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Defective viral genomes arisin ...... ing of lung antiviral immunity

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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type

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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Defective viral genomes arisin ...... ing of lung antiviral immunity

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Emily Dunay

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Karla Tapia

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Megan Wise

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Michael Adu

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Won-Keun Kim

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Xiomara Mercado-López

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Yan Sun

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P2860

Recognition of 5' triphosphate by RIG-I helicase requires short blunt double-stranded RNA as contained in panhandle of negative-strand virus.

The paramyxovirus, Sendai virus, V protein encodes a luxury function required for viral pathogenesis.

Sendai virus infection induces efficient adaptive immunity independently of type I interferons

The V proteins of paramyxoviruses bind the IFN-inducible RNA helicase, mda-5, and inhibit its activation of the IFN-beta promoter

Length-dependent recognition of double-stranded ribonucleic acids by retinoic acid-inducible gene-I and melanoma differentiation-associated gene 5

Characterization of Hepatitis C Virus Deletion Mutants Circulating in Chronically Infected Patients

Melanoma Differentiation-Associated Gene 5 (MDA5) Is Involved in the Innate Immune Response to Paramyxoviridae Infection In Vivo

RIG-I-mediated antiviral responses to single-stranded RNA bearing 5'-phosphates

5'-Triphosphate RNA is the ligand for RIG-I

Quasispecies diversity determines pathogenesis through cooperative interactions in a viral population

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e1003703

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10.1371/JOURNAL.PPAT.1003703

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10

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9

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Carolina B López

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258350526

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3814336

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