Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise. - Wikidata - wikimedia - TriplyDB

Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise.

Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise.

http://www.wikidata.org/entity/Q35035478

about

Non-coding RNAs: Epigenetic regulators of bone development and homeostasis.Genetic factors conferring metastasis in osteosarcoma.Mechanical Stress Regulates Bone Metabolism Through MicroRNAs.MicroRNAs in bone diseases.Mechanosensitive miRNAs and Bone Formation IGF-1 signaling mediated cell-specific skeletal mechano-transduction.Experimental repetitive mild traumatic brain injury induces deficits in trabecular bone microarchitecture and strength in mice.

P2860

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P2860

Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise.

http://www.wikidata.org/entity/Q35035478

description

2014 nî lūn-bûn

@nan

2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Conditional disruption of miR1 ...... anabolic response to exercise.

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Conditional disruption of miR1 ...... anabolic response to exercise.

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type

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Conditional disruption of miR1 ...... anabolic response to exercise.

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Conditional disruption of miR1 ...... anabolic response to exercise.

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Conditional disruption of miR1 ...... anabolic response to exercise.

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Conditional disruption of miR1 ...... anabolic response to exercise.

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PHYSIOLGENOMICS.00107.2014

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Physiological Genomics

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Conditional disruption of miR1 ...... anabolic response to exercise.

@en

P2093

Chandrasekhar Kesavan

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Jon E Wergedal

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Subburaman Mohan

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Subhashri Das

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P2860

Germline deletion of the miR-17∼92 cluster causes skeletal and growth defects in humans

Identification and characterization of a novel protein, periostin, with restricted expression to periosteum and periodontal ligament and increased expression by transforming growth factor beta

Disruption of insulin-like growth factor-I expression in type IIalphaI collagen-expressing cells reduces bone length and width in mice.

Ephrin B1 regulates bone marrow stromal cell differentiation and bone formation by influencing TAZ transactivation via complex formation with NHERF1.

Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse.

Phenotypic characterization of miR-92a-/- mice reveals an important function of miR-92a in skeletal development.

Conditional deletion of insulin-like growth factor-I in collagen type 1alpha2-expressing cells results in postnatal lethality and a dramatic reduction in bone accretion

miR-764-5p promotes osteoblast differentiation through inhibition of CHIP/STUB1 expression.

miR-214 targets ATF4 to inhibit bone formation.

Novel insights in the regulation and mechanism of androgen action on bone.

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33-43

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scholarly article

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10.1152/PHYSIOLGENOMICS.00107.2014

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2

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47

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