Cigarette smoke alters primary human bronchial epithelial cell differentiation at the air-liquid interface.
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Cigarette smoke alters the secretome of lung epithelial cells.Rapid Expansion of Human Epithelial Stem Cells Suitable for Airway Tissue Engineering.Impact Assessment of Cigarette Smoke Exposure on Organotypic Bronchial Epithelial Tissue Cultures: A Comparison of Mono-Culture and Coculture Model Containing Fibroblasts.Gefitinib, an EGFR Tyrosine Kinase inhibitor, Prevents Smoke-Mediated Ciliated Airway Epithelial Cell Loss and Promotes Their Recovery.Acetylated α-Tubulin Regulated by N-Acetyl-Seryl-Aspartyl-Lysyl-Proline(Ac-SDKP) Exerts the Anti-fibrotic Effect in Rat Lung Fibrosis Induced by SilicaDevelopment of Combining of Human Bronchial Mucosa Models with XposeALI® for Exposure of Air Pollution NanoparticlesHuman rhinovirus-induced inflammatory responses are inhibited by phosphatidylserine containing liposomesGlutathione peroxidase 3 localizes to the epithelial lining fluid and the extracellular matrix in interstitial lung disease.In Vitro Models to Study Human Lung Development, Disease and Homeostasis.Acute cigarette smoke exposure activates apoptotic and inflammatory programs but a second stimulus is required to induce epithelial to mesenchymal transition in COPD epithelium.Human papillomavirus and lung cancinogenesis: an overview.Immune-mediated inflammation in the pathogenesis of emphysema: insights from mouse models.Mitochondrial alterations during oxidative stress in chronic obstructive pulmonary disease.Human pulmonary 3D models for translational research.Cigarette smoke differentially affects IL-13-induced gene expression in human airway epithelial cellsDetection and quantification of epithelial progenitor cell populations in human healthy and IPF lungs.Autophagy plays an essential role in cigarette smoke-induced expression of MUC5AC in airway epithelium.Zinc deficiency as a codeterminant for airway epithelial barrier dysfunction in an ex vivo model of COPD.Exposure to cigarette smoke extract and lipopolysaccharide modifies cytoskeleton organization in bronchial epithelial cells.The uncoupling of autophagy and zinc homeostasis in airway epithelial cells as a fundamental contributor to COPD.Cadmium nanoparticles citrullinate cytokeratins within lung epithelial cells: cadmium as a potential cause of citrullination in chronic obstructive pulmonary disease.Mandatory role of HMGA1 in human airway epithelial normal differentiation and post-injury regeneration.Inhibition of the Hedgehog Signaling Pathway Depresses the Cigarette Smoke-Induced Malignant Transformation of 16HBE Cells on a Microfluidic Chip.
P2860
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P2860
Cigarette smoke alters primary human bronchial epithelial cell differentiation at the air-liquid interface.
description
2015 nî lūn-bûn
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2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Cigarette smoke alters primary ...... n at the air-liquid interface.
@ast
Cigarette smoke alters primary ...... n at the air-liquid interface.
@en
type
label
Cigarette smoke alters primary ...... n at the air-liquid interface.
@ast
Cigarette smoke alters primary ...... n at the air-liquid interface.
@en
prefLabel
Cigarette smoke alters primary ...... n at the air-liquid interface.
@ast
Cigarette smoke alters primary ...... n at the air-liquid interface.
@en
P2093
P2860
P356
P1433
P1476
Cigarette smoke alters primary ...... on at the air-liquid interface
@en
P2093
Andrea C Schamberger
Nikica Mise-Racek
Oliver Eickelberg
P2860
P2888
P356
10.1038/SREP08163
P407
P577
2015-02-02T00:00:00Z