Macrophage-derived tumor necrosis factor-alpha is an early component of the molecular cascade leading to angiogenesis in response to aortic injury
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MULTIMERIN2 impairs tumor angiogenesis and growth by interfering with VEGF-A/VEGFR2 pathwayAdipose Tissue Remodeling: Its Role in Energy Metabolism and Metabolic DisordersHif-1α regulates macrophage-endothelial interactions during blood vessel development in zebrafish.Downregulation of endothelial microRNA-200b supports cutaneous wound angiogenesis by desilencing GATA binding protein 2 and vascular endothelial growth factor receptor 2The acute phase reactant orosomucoid-1 is a bimodal regulator of angiogenesis with time- and context-dependent inhibitory and stimulatory propertiesKnockout of CD8 delays reendothelialization and accelerates neointima formation in injured arteries of mouse via TNF-α inhibiting the endothelial cells migration.Local delivery of polarized macrophages improves reperfusion recovery in a mouse hind limb ischemia model.Interleukin-19 increases angiogenesis in ischemic hind limbs by direct effects on both endothelial cells and macrophage polarization.Humanin prevents intra-renal microvascular remodeling and inflammation in hypercholesterolemic ApoE deficient mice.The role of vascular endothelial growth factor in metastatic prostate cancer to the skeleton.PEDF mediates pathological neovascularization by regulating macrophage recruitment and polarization in the mouse model of oxygen-induced retinopathy.High-mobility group box 1 protein activating nuclear factor-κB to upregulate vascular endothelial growth factor C is involved in lymphangiogenesis and lymphatic node metastasis in colon cancer.Identification of different macrophage subpopulations with distinct activities in a mouse model of oxygen-induced retinopathy.Interleukin-17A neutralization alleviated ocular neovascularization by promoting M2 and mitigating M1 macrophage polarization.Bone regeneration is associated with the concentration of tumour necrosis factor-α induced by sericin released from a silk mat.IL-17 in neonatal health and disease.Recruitment of macrophages from the spleen contributes to myocardial fibrosis and hypertension induced by angiotensin II.Hypoxia paradoxically inhibits the angiogenic response of isolated vessel explants while inducing overexpression of vascular endothelial growth factor.Hyperactive FOXO1 results in lack of tip stalk identity and deficient microvascular regeneration during kidney injury.M1 macrophages act as LTβR-independent lymphoid tissue inducer cells during atherosclerosis-related lymphoid neogenesis.VEGF-A/VEGFR-2 and FGF-2/FGFR-1 but not PDGF-BB/PDGFR-β play important roles in promoting immature and inflammatory intraplaque angiogenesis
P2860
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P2860
Macrophage-derived tumor necrosis factor-alpha is an early component of the molecular cascade leading to angiogenesis in response to aortic injury
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Macrophage-derived tumor necro ...... s in response to aortic injury
@ast
Macrophage-derived tumor necro ...... s in response to aortic injury
@en
type
label
Macrophage-derived tumor necro ...... s in response to aortic injury
@ast
Macrophage-derived tumor necro ...... s in response to aortic injury
@en
prefLabel
Macrophage-derived tumor necro ...... s in response to aortic injury
@ast
Macrophage-derived tumor necro ...... s in response to aortic injury
@en
P2093
P2860
P1476
Macrophage-derived tumor necro ...... s in response to aortic injury
@en
P2093
Alfred C Aplin
Ann Morishita
Giovanni Ligresti
Penelope Zorzi
Roberto F Nicosia
P2860
P304
P356
10.1161/ATVBAHA.111.223917
P407
P577
2011-03-03T00:00:00Z