Epidermal growth factor receptor tyrosine kinase-mediated signalling contributes to diabetes-induced vascular dysfunction in the mesenteric bed.

Epidermal growth factor receptor tyrosine kinase-mediated signalling contributes to diabetes-induced vascular dysfunction in the mesenteric bed.

Item
http://www.wikidata.org/entity/Q35049004
Expression-based network biology identifies alteration in key regulatory pathways of type 2 diabetes and associated risk/complicationsCationic Polyamidoamine Dendrimers as Modulators of EGFR Signaling In Vitro and In VivoActivation of EGFR/ERBB2 via pathways involving ERK1/2, P38 MAPK, AKT and FOXO enhances recovery of diabetic hearts from ischemia-reperfusion injuryTransactivation of ErbB Family of Receptor Tyrosine Kinases Is Inhibited by Angiotensin-(1-7) via Its Mas Receptor.Activation of ErbB2 and Downstream Signalling via Rho Kinases and ERK1/2 Contributes to Diabetes-Induced Vascular Dysfunction.Advanced glycation end product (AGE) receptor 1 suppresses cell oxidant stress and activation signaling via EGF receptorAngiotensin-(1-7) inhibits epidermal growth factor receptor transactivation via a Mas receptor-dependent pathway.Nuclear factor kappa B inhibition improves conductance artery function in type 2 diabetic miceHerbal therapies for type 2 diabetes mellitus: chemistry, biology, and potential application of selected plants and compoundsAugmented EGF receptor tyrosine kinase activity impairs vascular function by NADPH oxidase-dependent mechanism in type 2 diabetic mouseRU28318, an aldosterone antagonist, in combination with an ACE inhibitor and angiotensin receptor blocker attenuates cardiac dysfunction in diabetes.The epidermal growth factor receptor and its ligands in cardiovascular disease.A novel role for epidermal growth factor receptor tyrosine kinase and its downstream endoplasmic reticulum stress in cardiac damage and microvascular dysfunction in type 1 diabetes mellitus.The role of epidermal growth factor receptor in diabetes-induced cardiac dysfunction.Src-dependent EGFR transactivation regulates lung inflammation via downstream signaling involving ERK1/2, PI3Kδ/Akt and NFκB induction in a murine asthma model.The activation of G protein-coupled estrogen receptor induces relaxation via cAMP as well as potentiates contraction via EGFR transactivation in porcine coronary arteries.The dual targeting of EGFR and ErbB2 with the inhibitor Lapatinib corrects high glucose-induced apoptosis and vascular dysfunction by opposing multiple diabetes-induced signaling changes.
P2860

Epidermal growth factor receptor tyrosine kinase-mediated signalling contributes to diabetes-induced vascular dysfunction in the mesenteric bed.

Item
http://www.wikidata.org/entity/Q35049004
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Epidermal growth factor recept ...... unction in the mesenteric bed.
@ast
Epidermal growth factor recept ...... unction in the mesenteric bed.
@en
type
label
Epidermal growth factor recept ...... unction in the mesenteric bed.
@ast
Epidermal growth factor recept ...... unction in the mesenteric bed.
@en
prefLabel
Epidermal growth factor recept ...... unction in the mesenteric bed.
@ast
Epidermal growth factor recept ...... unction in the mesenteric bed.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P698
P932
P356
P1433
P1476
Epidermal growth factor recept ...... function in the mesenteric bed
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P698
P932