Acute lung injury and acute kidney injury are established by four hours in experimental sepsis and are improved with pre, but not post, sepsis administration of TNF-α antibodies.
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Why do they die? Comparison of selected aspects of organ injury and dysfunction in mice surviving and dying in acute abdominal sepsisUrinary metabolomics of bronchopulmonary dysplasia (BPD): preliminary data at birth suggest it is a congenital disease.Blockade of ICAM-1 improves the outcome of polymicrobial sepsis via modulating neutrophil migration and reversing immunosuppression.Suppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury.Current Murine Models of Sepsis.Polydatin attenuates ipopolysaccharide-induced acute lung injury in rats.Cecal ligation and puncture accelerates development of ventilator-induced lung injury.Myeloperoxidase formation of PAF receptor ligands induces PAF receptor-dependent kidney injury during ethanol consumptionEnhanced Resolution of Hyperoxic Acute Lung Injury as a result of Aspirin Triggered Resolvin D1 Treatment.Peripheral Organs of Dengue Fatal Cases Present Strong Pro-Inflammatory Response with Participation of IFN-Gamma-, TNF-Alpha- and RANTES-Producing Cells.The long noncoding RNA landscape in hypoxic and inflammatory renal epithelial injury.Vitamin D3 pretreatment regulates renal inflammatory responses during lipopolysaccharide-induced acute kidney injury.Bridging translation for acute kidney injury with better preclinical modeling of human diseaseHeparin-binding protein is important for vascular leak in sepsisEffect of endotoxemia in mice genetically deficient in cystathionine-γ-lyase, cystathionine-β-synthase or 3-mercaptopyruvate sulfurtransferase.Mechanisms and mediators of lung injury after acute kidney injury.Early peritoneal dialysis reduces lung inflammation in mice with ischemic acute kidney injury.A model-specific role of microRNA-223 as a mediator of kidney injury during experimental sepsis.Spleen contributes significantly to increased circulating levels of fibroblast growth factor 23 in response to lipopolysaccharide-induced inflammation.Can neutrophil-lymphocyte ratio be independent risk factor for predicting acute kidney injury in patients with severe sepsis?Gingerol suppresses sepsis-induced acute kidney injury by modulating methylsulfonylmethane and dimethylamine production
P2860
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P2860
Acute lung injury and acute kidney injury are established by four hours in experimental sepsis and are improved with pre, but not post, sepsis administration of TNF-α antibodies.
description
2013 nî lūn-bûn
@nan
2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@ast
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@en
type
label
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@ast
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@en
prefLabel
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@ast
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@en
P2093
P2860
P1433
P1476
Acute lung injury and acute ki ...... istration of TNF-α antibodies.
@en
P2093
Ana Andres-Hernando
Christopher J Altmann
Kayo Okamura
Rhea Bhargava
Ryan G Webb
Sandor Falk
Sarah Faubel
P2860
P304
P356
10.1371/JOURNAL.PONE.0079037
P407
P577
2013-11-12T00:00:00Z