Inhibition of transforming growth factor beta/SMAD signal by MiR-155 is involved in arsenic trioxide-induced anti-angiogenesis in prostate cancer.

Inhibition of transforming growth factor beta/SMAD signal by MiR-155 is involved in arsenic trioxide-induced anti-angiogenesis in prostate cancer.

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http://www.wikidata.org/entity/Q35052004
Blockage of TGFβ-SMAD2 by demethylation-activated miR-148a is involved in caffeic acid-induced inhibition of cancer stem cell-like properties in vitro and in vivo.Increased duodenal expression of miR-146a and -155 in pediatric Crohn's disease.MicroRNA-155 deletion promotes tumorigenesis in the azoxymethane-dextran sulfate sodium model of colon cancer.Induction of the mesenchymal to epithelial transition by demethylation-activated microRNA-125b is involved in the anti-migration/invasion effects of arsenic trioxide on human chondrosarcoma.U937 cell apoptosis induced by arsenite is prevented by low concentrations of mitochondrial ascorbic acid with hardly any effect mediated by the cytosolic fraction of the vitamin.miRNAs in Normal and Malignant Hematopoiesis.Anti-angiogenic effect of arsenic trioxide in lung cancer via inhibition of endothelial cell migration, proliferation and tube formation.Arsenic trioxide increases expression of secreted frizzled-related protein 1 gene and inhibits the WNT/β-catenin signaling pathway in Jurkat cells.miR-155 mediates arsenic trioxide resistance by activating Nrf2 and suppressing apoptosis in lung cancer cells.Dihydroartemisinin Sensitizes Human Lung Adenocarcinoma A549 Cells to Arsenic Trioxide via Apoptosis.Inhibition of STAT3/VEGF/CDK2 axis signaling is critically involved in the antiangiogenic and apoptotic effects of arsenic herbal mixture PROS in non-small lung cancer cells.Clinical significance of miRNA host gene promoter methylation in prostate cancer.Prostate-specific PTen deletion in mice activates inflammatory microRNA expression pathways in the epithelium early in hyperplasia development.Arsenic trioxide inhibits cell growth and motility via up-regulation of let-7a in breast cancer cells.The association of plasma miR-155 and VCAM-1 levels with coronary collateral circulation.2-Methoxyestradiol Attenuates Testosterone-Induced Benign Prostate Hyperplasia in Rats through Inhibition of HIF-1/TGF-/Smad2 Axis
P2860

Inhibition of transforming growth factor beta/SMAD signal by MiR-155 is involved in arsenic trioxide-induced anti-angiogenesis in prostate cancer.

Item
http://www.wikidata.org/entity/Q35052004
description
2014 nî lūn-bûn
@nan
2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@ast
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@en
type
label
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@ast
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@en
prefLabel
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@ast
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P932
P356
P1433
P1476
Inhibition of transforming gro ...... giogenesis in prostate cancer.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P932