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Emerging therapeutic targets in chronic heart failure: part II.

Emerging therapeutic targets in chronic heart failure: part II.

http://www.wikidata.org/entity/Q35056026

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Myocyte enhancer factor 2 and class II histone deacetylases control a gender-specific pathway of cardioprotection mediated by the estrogen receptor.Small molecule and peptide therapies for chronic heart failure: a patent review (2011 - 2014).

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Emerging therapeutic targets in chronic heart failure: part II.

http://www.wikidata.org/entity/Q35056026

description

2003 nî lūn-bûn

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2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի փետրվարին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Emerging therapeutic targets in chronic heart failure: part II.

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Emerging therapeutic targets in chronic heart failure: part II.

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type

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Emerging therapeutic targets in chronic heart failure: part II.

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Emerging therapeutic targets in chronic heart failure: part II.

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Emerging therapeutic targets in chronic heart failure: part II.

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Emerging therapeutic targets in chronic heart failure: part II.

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14728222.7.1.49

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Expert Opinion on Therapeutic Targets

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Emerging therapeutic targets in chronic heart failure: part II.

@en

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Anke Hoffmann

http://www.w3.org/2001/XMLSchema#string

Barbara T Navé

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Michael Becker

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Oliver Stoss

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Stéphane Leclair

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Tania Szado

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Thomas Henkel

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Thomas Kirsch

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Volker Roenicke

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P2860

Akt phosphorylates and negatively regulates apoptosis signal-regulating kinase 1

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

Expression cloning of cardiotrophin 1, a cytokine that induces cardiac myocyte hypertrophy

Requirement of ErbB2 for signalling by interleukin-6 in prostate carcinoma cells

Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

Bone marrow cells regenerate infarcted myocardium

Aging, cardiac hypertrophy and ischemic cardiomyopathy do not affect the proportion of mononucleated and multinucleated myocytes in the human heart

Cardiac function in mice overexpressing the beta-adrenergic receptor kinase or a beta ARK inhibitor

Role of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophy

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49-59

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