LPS exposure increases maternal corticosterone levels, causes placental injury and increases IL-1Β levels in adult rat offspring: relevance to autism.
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Molecular substrates of schizophrenia: homeostatic signaling to connectivityAstaxanthin ameliorates prenatal LPS-exposed behavioral deficits and oxidative stress in adult offspring.Maternal obesity, inflammation, and developmental programming.Cytokine profiles by peripheral blood monocytes are associated with changes in behavioral symptoms following immune insults in a subset of ASD subjects: an inflammatory subtype?Effects of maternal LPS exposure during pregnancy on metabolic phenotypes in female offspringZinc prevents sickness behavior induced by lipopolysaccharides after a stress challenge in rats.Lipopolysaccharide Exposure Induces Maternal Hypozincemia, and Prenatal Zinc Treatment Prevents Autistic-Like Behaviors and Disturbances in the Striatal Dopaminergic and mTOR Systems of OffspringPropentofylline Prevents Sickness Behavior and Depressive-Like Behavior Induced by Lipopolysaccharide in Rats via Neuroinflammatory PathwayAutism biomarkers: challenges, pitfalls and possibilities.Maternal Lipopolysaccharide Exposure Promotes Immunological Functional Changes in Adult Offspring CD4+ T Cells.Neuroteratology and Animal Modeling of Brain Disorders.Prenatal fever and autism risk.The Role of C5a Receptor Signaling in Endotoxin-Induced Miscarriage and Preterm Birth.The impact of maternal obesity on inflammatory processes and consequences for later offspring health outcomes.A sexually dichotomous, autistic-like phenotype is induced by Group B Streptococcus maternofetal immune activation.Neonatal Immune Challenge with Lipopolysaccharide Triggers Long-lasting Sex- and Age-related Behavioral and Immune/Neurotrophic Alterations in Mice: Relevance to Autism Spectrum Disorders.Maternal immune activation produces cerebellar hyperplasia and alterations in motor and social behaviors in male and female mice.Positive modulation of α5 GABAA receptors in preadolescence prevents reduced locomotor response to amphetamine in adult female but not male rats prenatally exposed to lipopolysaccharide.Maternal immune activation in neurodevelopmental disorders.Toll-like receptor 4-mediated immune stress in pregnant rats activates STAT3 in the fetal brain: role of interleukin-6.Disruptions in the hypothalamic-pituitary-gonadal axis in rat offspring following prenatal maternal exposure to lipopolysaccharide.Pioglitazone abolishes autistic-like behaviors via the IL-6 pathway.p38α MAPK signaling drives pharmacologically reversible brain and gastrointestinal phenotypes in the SERT Ala56 mouse
P2860
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P2860
LPS exposure increases maternal corticosterone levels, causes placental injury and increases IL-1Β levels in adult rat offspring: relevance to autism.
description
2013 nî lūn-bûn
@nan
2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
LPS exposure increases materna ...... ffspring: relevance to autism.
@ast
LPS exposure increases materna ...... ffspring: relevance to autism.
@en
type
label
LPS exposure increases materna ...... ffspring: relevance to autism.
@ast
LPS exposure increases materna ...... ffspring: relevance to autism.
@en
prefLabel
LPS exposure increases materna ...... ffspring: relevance to autism.
@ast
LPS exposure increases materna ...... ffspring: relevance to autism.
@en
P2860
P50
P1433
P1476
LPS exposure increases materna ...... ffspring: relevance to autism.
@en
P2093
Luciana L Lippi
P2860
P304
P356
10.1371/JOURNAL.PONE.0082244
P407
P577
2013-12-02T00:00:00Z