Peripheral blood CCR4+CCR6+ and CXCR3+CCR6+CD4+ T cells are highly permissive to HIV-1 infection. - Wikidata - wikimedia - TriplyDB

Peripheral blood CCR4+CCR6+ and CXCR3+CCR6+CD4+ T cells are highly permissive to HIV-1 infection.

Peripheral blood CCR4+CCR6+ and CXCR3+CCR6+CD4+ T cells are highly permissive to HIV-1 infection.

http://www.wikidata.org/entity/Q35064033

about

Asymmetric HIV-1 co-receptor use and replication in CD4(+) T lymphocytes HIV-1-specific interleukin-21+ CD4+ T cell responses contribute to durable viral control through the modulation of HIV-specific CD8+ T cell function Gut barrier structure, mucosal immunity and intestinal microbiota in the pathogenesis and treatment of HIV infection New Insights about Treg and Th17 Cells in HIV Infection and Disease Progression Oropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 Transgene Long-term programming of antigen-specific immunity from gene expression signatures in the PBMC of rhesus macaques immunized with an SIV DNA vaccine CCR5/CD4/CXCR4 oligomerization prevents HIV-1 gp120IIIB binding to the cell surface Plasma IP-10 Concentrations Correlate Positively with Viraemia and Inversely with CD4 Counts in Untreated HIV Infection Human Beta Defensin 2 Selectively Inhibits HIV-1 in Highly Permissive CCR6⁺CD4⁺ T Cells.HIV persistence: chemokines and their signalling pathways.The loss of CCR6+ and CD161+ CD4+ T-cell homeostasis contributes to disease progression in SIV-infected rhesus macaques.Differences in serum IgA responses to HIV-1 gp41 in elite controllers compared to viral suppressors on highly active antiretroviral therapy Transcriptional profile of tuberculosis antigen-specific T cells reveals novel multifunctional features The colocalization potential of HIV-specific CD8+ and CD4+ T-cells is mediated by integrin β7 but not CCR6 and regulated by retinoic acid.Loss and dysregulation of Th17 cells during HIV infection.Phenotype and susceptibility to HIV infection of CD4+ Th17 cells in the human female reproductive tract.Blunted IL17/IL22 and pro-inflammatory cytokine responses in the genital tract and blood of HIV-exposed, seronegative female sex workers in Kenya Defective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.Antibody persistence and T-cell balance: two key factors confronting HIV vaccine development.Impact of asymptomatic Herpes simplex virus-2 infection on T cell phenotype and function in the foreskin.The Th17/Treg ratio, IL-1RA and sCD14 levels in primary HIV infection predict the T-cell activation set point in the absence of systemic microbial translocation.CCR6 functions as a new coreceptor for limited primary human and simian immunodeficiency viruses.Low SAMHD1 expression following T-cell activation and proliferation renders CD4+ T cells susceptible to HIV-1.CD4+ T cells from elite suppressors are more susceptible to HIV-1 but produce fewer virions than cells from chronic progressors.Site-specific differences in T cell frequencies and phenotypes in the blood and gut of HIV-uninfected and ART-treated HIV+ adults.Impaired Th17 polarization of phenotypically naive CD4(+) T-cells during chronic HIV-1 infection and potential restoration with early ART TFH cells accumulate in mucosal tissues of humanized-DRAG mice and are highly permissive to HIV-1 Schistosoma mansoni Infection in Ugandan Men Is Associated with Increased Abundance and Function of HIV Target Cells in Blood, but Not the Foreskin: A Cross-sectional Study.Foreskin T-cell subsets differ substantially from blood with respect to HIV co-receptor expression, inflammatory profile, and memory status Integrin α4β7 Expression Increases HIV Susceptibility in Activated Cervical CD4+ T Cells by an HIV Attachment-Independent Mechanism.Codelivery of Envelope Protein in Alum with MVA Vaccine Induces CXCR3-Biased CXCR5+ and CXCR5- CD4 T Cell Responses in Rhesus Macaques.Selective Loss of Early Differentiated, Highly Functional PD1high CD4 T Cells with HIV Progression.Chemokine Receptor Ccr6 Deficiency Alters Hepatic Inflammatory Cell Recruitment and Promotes Liver Inflammation and Fibrosis.Rapid and Slow Progressors Show Increased IL-6 and IL-10 Levels in the Pre-AIDS Stage of HIV Infection.CD4+ T Cells Expressing PD-1, TIGIT and LAG-3 Contribute to HIV Persistence during ART.HIV-1 infection of human intestinal lamina propria CD4+ T cells in vitro is enhanced by exposure to commensal Escherichia coli.Elevated Basal Pre-infection CXCL10 in Plasma and in the Small Intestine after Infection Are Associated with More Rapid HIV/SIV Disease Onset HIV persistence in the setting of antiretroviral therapy: when, where and how does HIV hide?Increased susceptibility of CD4+ T cells from elderly individuals to HIV-1 infection and apoptosis is associated with reduced CD4 and enhanced CXCR4 and FAS surface expression levels Glycosylation of simian immunodeficiency virus influences immune-tissue targeting during primary infection, leading to immunodeficiency or viral control

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P2860

Peripheral blood CCR4+CCR6+ and CXCR3+CCR6+CD4+ T cells are highly permissive to HIV-1 infection.

http://www.wikidata.org/entity/Q35064033

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

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Peripheral blood CCR4+CCR6+ an ...... permissive to HIV-1 infection.

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JIMMUNOL.0903058

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Journal of Immunology

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Peripheral blood CCR4+CCR6+ an ...... permissive to HIV-1 infection.

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P2093

Annie Gosselin

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Elias A Said

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Felipe Diaz-Griffero

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Jean-Pierre Routy

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Mohamed El-Far

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Mohamed-Rachid Boulassel

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Patricia Monteiro

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Petronela Ancuta

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Simone Fonseca

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Vanessa Wacleche

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P2860

Beta-defensins: linking innate and adaptive immunity through dendritic and T cell CCR6

CD4+ T cell depletion during all stages of HIV disease occurs predominantly in the gastrointestinal tract

Primary HIV-1 infection is associated with preferential depletion of CD4+ T lymphocytes from effector sites in the gastrointestinal tract

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

HIV-1 envelope protein binds to and signals through integrin α4β7, the gut mucosal homing receptor for peripheral T cells

A new classification for HIV-1

The lymphocyte chemoattractant SDF-1 is a ligand for LESTR/fusin and blocks HIV-1 entry

Critical loss of the balance between Th17 and T regulatory cell populations in pathogenic SIV infection

Two subsets of memory T lymphocytes with distinct homing potentials and effector functions

The challenge of viral reservoirs in HIV-1 infection

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1604-1616

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scholarly article

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10.4049/JIMMUNOL.0903058

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3

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184

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Nicolas Chomont

Rafick-Pierre Sekaly

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2009-12-30T00:00:00Z

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40819573

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20042588

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4321756

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