Chloride channel (Clc)-5 is necessary for exocytic trafficking of Na+/H+ exchanger 3 (NHE3). - Wikidata - wikimedia - TriplyDB

Chloride channel (Clc)-5 is necessary for exocytic trafficking of Na+/H+ exchanger 3 (NHE3).

Chloride channel (Clc)-5 is necessary for exocytic trafficking of Na+/H+ exchanger 3 (NHE3).

http://www.wikidata.org/entity/Q35067687

about

Molecular mechanisms and regulation of urinary acidification Protein trafficking defects in inherited kidney diseases WDR72 models of structure and function: a stage-specific regulator of enamel mineralization The Lowe syndrome protein OCRL1 is required for endocytosis in the zebrafish pronephric tubule.Sorting nexin 27 regulates basal and stimulated brush border trafficking of NHE3.The CLC-5 2Cl(-)/H(+) exchange transporter in endosomal function and Dent's disease.Chloride channel ClC-5 binds to aspartyl aminopeptidase to regulate renal albumin endocytosis Barttin Regulates the Subcellular Localization and Posttranslational Modification of Human Cl/H Antiporter ClC-5

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Chloride channel (Clc)-5 is necessary for exocytic trafficking of Na+/H+ exchanger 3 (NHE3).

http://www.wikidata.org/entity/Q35067687

description

2011 nî lūn-bûn

@nan

2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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type

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Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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prefLabel

Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

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JBC.M111.224998

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Journal of Biological Chemistry

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Chloride channel (Clc)-5 is ne ...... of Na+/H+ exchanger 3 (NHE3).

@en

P2093

Ann Hubbard

http://www.w3.org/2001/XMLSchema#string

Boyoung Cha

http://www.w3.org/2001/XMLSchema#string

Mark Donowitz

http://www.w3.org/2001/XMLSchema#string

Phuson Hulamm

http://www.w3.org/2001/XMLSchema#string

Sabrina Martini

http://www.w3.org/2001/XMLSchema#string

Sandra E Guggino

http://www.w3.org/2001/XMLSchema#string

Shi Jin

http://www.w3.org/2001/XMLSchema#string

Tong Wang

http://www.w3.org/2001/XMLSchema#string

Xiaohong Duan

http://www.w3.org/2001/XMLSchema#string

Zhihong Lin

http://www.w3.org/2001/XMLSchema#string

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Idiopathic low molecular weight proteinuria associated with hypercalciuric nephrocalcinosis in Japanese children is due to mutations of the renal chloride channel (CLCN5)

cAMP-mediated inhibition of the epithelial brush border Na+/H+ exchanger, NHE3, requires an associated regulatory protein

ClC-5, the chloride channel mutated in Dent's disease, colocalizes with the proton pump in endocytotically active kidney cells

The CLC chloride channel family

Glucocorticoid activation of Na(+)/H(+) exchanger isoform 3 revisited. The roles of SGK1 and NHERF2

High citrate diet delays progression of renal insufficiency in the ClC-5 knockout mouse model of Dent's disease

Na+-H+ exchanger 3 (NHE3) is present in lipid rafts in the rabbit ileal brush border: a role for rafts in trafficking and rapid stimulation of NHE3

Intrarenal and subcellular localization of rat CLC5

A chloride channel widely expressed in epithelial and non-epithelial cells

Loss of chloride channel ClC-5 impairs endocytosis by defective trafficking of megalin and cubilin in kidney proximal tubules

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22833-22845

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scholarly article

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10.1074/JBC.M111.224998

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26

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286

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2011-05-11T00:00:00Z

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21561868

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3123051

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