Pathogenesis of multiple organ dysfunction syndrome--endotoxin, inflammatory cells, and their mediators: cytokines and reactive oxygen species.

Pathogenesis of multiple organ dysfunction syndrome--endotoxin, inflammatory cells, and their mediators: cytokines and reactive oxygen species.

Item
http://www.wikidata.org/entity/Q35069248
Clinical, haematological and biochemical responses of sheep undergoing autologous blood transfusion.The role of intestinal mucosa injury induced by intra-abdominal hypertension in the development of abdominal compartment syndrome and multiple organ dysfunction syndrome.Pancreatic trypsin increases matrix metalloproteinase-9 accumulation and activation during acute intestinal ischemia-reperfusion in the rat.Translational systems approaches to the biology of inflammation and healing.Sepsis: Something old, something new, and a systems view.Evidence-based modeling of critical illness: an initial consensus from the Society for Complexity in Acute Illness.Impact of off-pump coronary artery bypass surgery on systemic inflammation: current best available evidence.Calcium supplementation during sepsis exacerbates organ failure and mortality via calcium/calmodulin-dependent protein kinase kinase signalingDeletion of Nlrp3 Augments Survival during Polymicrobial Sepsis by Decreasing Autophagy and Enhancing Phagocytosis.Reduced oxidative tissue damage during endotoxemia in IRAK-1 deficient miceHypertonic saline resuscitation maintains a more balanced profile of T-lymphocyte subpopulations in a rat model of hemorrhagic shock.Cytochrome c modulates the mitochondrial signaling pathway and polymorphonuclear neutrophil apoptosis in bile duct-ligated rats.Natural Hirudin Increases Rat Flap Viability by Anti-Inflammation via PARs/p38/NF-κB Pathway.Pentoxifylline modulates p47phox activation and downregulates neutrophil oxidative burst through PKA-dependent and -independent mechanisms.Cool seasons are related to poor prognosis in patients with infective endocarditis.Hypertonic resuscitation modulates the inflammatory response in patients with traumatic hemorrhagic shock.Beta2-microglobulin knockout mice treated with anti-asialoGM1 exhibit improved hemodynamics and cardiac contractile function during acute intra-abdominal sepsis.
P2860

Pathogenesis of multiple organ dysfunction syndrome--endotoxin, inflammatory cells, and their mediators: cytokines and reactive oxygen species.

Item
http://www.wikidata.org/entity/Q35069248
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@ast
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@en
type
label
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@ast
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@en
prefLabel
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@ast
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@en
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P921
P356
P1433
P1476
Pathogenesis of multiple organ ...... s and reactive oxygen species.
@en
P2093
P304
P31
P356
10.1089/109629600750018123
P433
P478
P577
P698
P921