Different consequences of ACE2 and SWI5 gene disruptions for virulence of pathogenic and nonpathogenic yeasts. - Wikidata - wikimedia - TriplyDB

Different consequences of ACE2 and SWI5 gene disruptions for virulence of pathogenic and nonpathogenic yeasts.

Different consequences of ACE2 and SWI5 gene disruptions for virulence of pathogenic and nonpathogenic yeasts.

http://www.wikidata.org/entity/Q35073747

about

Candida glabrata: new tools and technologies-expanding the toolkit Gain of function mutations in CgPDR1 of Candida glabrata not only mediate antifungal resistance but also enhance virulence One small step for a yeast--microevolution within macrophages renders Candida glabrata hypervirulent due to a single point mutation Transcriptional Control of Drug Resistance, Virulence and Immune System Evasion in Pathogenic Fungi: A Cross-Species Comparison Regulation of hypoxia adaptation: an overlooked virulence attribute of pathogenic fungi?Differential virulence of Candida glabrata glycosylation mutants Regulatory circuitry governing fungal development, drug resistance, and disease.Hosting infection: experimental models to assay Candida virulence The RAM network in pathogenic fungi.Coordination of hypoxia adaptation and iron homeostasis in human pathogenic fungi.The Pneumocystis Ace2 transcription factor regulates cell wall-remodeling genes and organism virulence.Property differences among the four major Candida albicans strain clades The Aspergillus fumigatus transcription factor Ace2 governs pigment production, conidiation and virulence.Candida albicans transcription factor Rim101 mediates pathogenic interactions through cell wall functions.Genome duplication and mutations in ACE2 cause multicellular, fast-sedimenting phenotypes in evolved Saccharomyces cerevisiae.From Saccharomyces cerevisiae to Candida glabratain a few easy steps: important adaptations for an opportunistic pathogen.Intracellular survival of Candida glabrata in macrophages: immune evasion and persistence.Evolution of regulatory networks in Candida glabrata: learning to live with the human host.The birth of a deadly yeast: tracing the evolutionary emergence of virulence traits in Candida glabrata.Candida albicans and Candida dubliniensis respond differently to echinocandin antifungal agents in vitro.The ATP-binding cassette transporter-encoding gene CgSNQ2 is contributing to the CgPDR1-dependent azole resistance of Candida glabrata.Pathogenesis and Antifungal Drug Resistance of the Human Fungal Pathogen Candida glabrata.Candida Biofilms: Threats, Challenges, and Promising Strategies.

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P2860

Different consequences of ACE2 and SWI5 gene disruptions for virulence of pathogenic and nonpathogenic yeasts.

http://www.wikidata.org/entity/Q35073747

description

2006 nî lūn-bûn

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2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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type

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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Infection and Immunity

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Different consequences of ACE2 ...... enic and nonpathogenic yeasts.

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P2093

Claire C Kenny

http://www.w3.org/2001/XMLSchema#string

Frank C Odds

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Helen Findon

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Ken Haynes

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P2860

Genome evolution in yeasts

The Saccharomyces cerevisiae Mob2p-Cbk1p kinase complex promotes polarized growth and acts with the mitotic exit network to facilitate daughter cell-specific localization of Ace2p transcription factor

Yeast Cbk1 and Mob2 activate daughter-specific genetic programs to induce asymmetric cell fates.

Overlapping and distinct roles of the duplicated yeast transcription factors Ace2p and Swi5p.

New heterologous modules for classical or PCR-based gene disruptions in Saccharomyces cerevisiae

Development of Saccharomyces cerevisiae as a model pathogen. A system for the genetic identification of gene products required for survival in the mammalian host environment

Transformation of yeast by lithium acetate/single-stranded carrier DNA/polyethylene glycol method

Serial regulation of transcriptional regulators in the yeast cell cycle

Candida orthopsilosis and Candida metapsilosis spp. nov. to replace Candida parapsilosis groups II and III.

Genetic characterization of pathogenic Saccharomyces cerevisiae isolates.

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5244-5248

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scholarly article

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10.1128/IAI.00817-06

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9

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74

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Geraldine Butler

Donna M MacCallum

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2006-09-01T00:00:00Z

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16926418

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1594847

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