Yersinia pestis YopJ suppresses tumor necrosis factor alpha induction and contributes to apoptosis of immune cells in the lymph node but is not required for virulence in a rat model of bubonic plague
about
Cell death programs in Yersinia immunity and pathogenesisYersinia type III effectors perturb host innate immune responsesIntegrating high-content imaging and chemical genetics to probe host cellular pathways critical for Yersinia pestis infectionNew insights into how Yersinia pestis adapts to its mammalian host during bubonic plagueOmics strategies for revealing Yersinia pestis virulenceMacrophage activation redirects yersinia-infected host cell death from apoptosis to caspase-1-dependent pyroptosis.Reduced secretion of YopJ by Yersinia limits in vivo cell death but enhances bacterial virulence.Yersinia pestis endowed with increased cytotoxicity is avirulent in a bubonic plague model and induces rapid protection against pneumonic plague.CD8(+) T cells restrict Yersinia pseudotuberculosis infection: bypass of anti-phagocytosis by targeting antigen-presenting cellsNeutrophils are resistant to Yersinia YopJ/P-induced apoptosis and are protected from ROS-mediated cell death by the type III secretion systemGlutathionylation of Yersinia pestis LcrV and Its Effects on Plague PathogenesisYopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infectionTn5AraOut mutagenesis for the identification of Yersinia pestis genes involved in resistance towards cationic antimicrobial peptidesA Yersinia effector protein promotes virulence by preventing inflammasome recognition of the type III secretion system.Novel plasmids and resistance phenotypes in Yersinia pestis: unique plasmid inventory of strain Java 9 mediates high levels of arsenic resistanceTranscriptomic and innate immune responses to Yersinia pestis in the lymph node during bubonic plagueBacterial programming of host responses: coordination between type I interferon and cell death.How the structural gene products of Yersinia pestis relate to virulence.Early apoptosis of macrophages modulated by injection of Yersinia pestis YopK promotes progression of primary pneumonic plagueCircumventing Y. pestis Virulence by Early Recruitment of Neutrophils to the Lungs during Pneumonic Plague.Insight into bacterial virulence mechanisms against host immune response via the Yersinia pestis-human protein-protein interaction network.Type III secretion system-dependent translocation of ectopically expressed Yop effectors into macrophages by intracellular Yersinia pseudotuberculosis.Growth of Yersinia pseudotuberculosis in mice occurs independently of Toll-like receptor 2 expression and induction of interleukin-10.Amino acid substitutions in LcrV at putative sites of interaction with Toll-like receptor 2 do not affect the virulence of Yersinia pestisRole of the Yersinia YopJ protein in suppressing interleukin-8 secretion by human polymorphonuclear leukocytes.FGL chaperone-assembled fimbrial polyadhesins: anti-immune armament of Gram-negative bacterial pathogens.Manipulation of Interleukin-1β and Interleukin-18 Production by Yersinia pestis Effectors YopJ and YopM and Redundant Impact on Virulence.Regulatory mechanisms underlying sepsis progression in patients with tumor necrosis factor-α genetic variationsPneumonic plague pathogenesis and immunity in Brown Norway rats.Modulation of host immune defenses by Aeromonas and Yersinia species: convergence on toxins secreted by various secretion systems.The cytotoxic necrotizing factor of Yersinia pseudotuberculosis (CNFY) enhances inflammation and Yop delivery during infection by activation of Rho GTPases.Gr1+ cells control growth of YopM-negative yersinia pestis during systemic plague.Innate immune response during Yersinia infection: critical modulation of cell death mechanisms through phagocyte activation.Murine neonates infected with Yersinia enterocolitica develop rapid and robust proinflammatory responses in intestinal lymphoid tissuesAdhesive organelles of Gram-negative pathogens assembled with the classical chaperone/usher machinery: structure and function from a clinical standpoint.Host Defense and the Airway Epithelium: Frontline Responses That Protect against Bacterial Invasion and Pneumonia.Activation and Evasion of Inflammasomes by Yersinia.Pathology and Pathogenesis of Yersinia pestis.Kinetics of innate immune response to Yersinia pestis after intradermal infection in a mouse model.Detection and isolation of Yersinia pestis without fraction 1 antigen by monoclonal antibody in foods and water.
P2860
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P2860
Yersinia pestis YopJ suppresses tumor necrosis factor alpha induction and contributes to apoptosis of immune cells in the lymph node but is not required for virulence in a rat model of bubonic plague
description
2006 nî lūn-bûn
@nan
2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@ast
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@en
type
label
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@ast
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@en
prefLabel
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@ast
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@en
P2860
P356
P1476
Yersinia pestis YopJ suppresse ...... a rat model of bubonic plague
@en
P2093
B Joseph Hinnebusch
Daniel Long
P2860
P304
P356
10.1128/IAI.00219-06
P407
P577
2006-09-01T00:00:00Z