PhoP-induced genes within Salmonella pathogenicity island 1.
about
Defining the plasticity of transcription factor binding sites by Deconstructing DNA consensus sequences: the PhoP-binding sites among gamma/enterobacteria.Pathogenicity of Salmonella enterica in Caenorhabditis elegans relies on disseminated oxidative stress in the infected hostSerratia marcescens is able to survive and proliferate in autophagic-like vacuoles inside non-phagocytic cellsSalmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Temperature and Mg2+ sensing by a novel PhoP-PhoQ two-component system for regulation of virulence in Edwardsiella tarda.Patterns of expression and translocation of the ubiquitin ligase SlrP in Salmonella enterica serovar Typhimurium.The promoter architectural landscape of the Salmonella PhoP regulon.The Xanthomonas oryzae pv. oryzae PhoPQ two-component system is required for AvrXA21 activity, hrpG expression, and virulence.Delineation of the Salmonella enterica serovar Typhimurium HilA regulon through genome-wide location and transcript analysisPotassium transport of Salmonella is important for type III secretion and pathogenesis.mgtA Expression is induced by rob overexpression and mediates a Salmonella enterica resistance phenotypeTargeting of DC-SIGN on human dendritic cells by minor fimbriated Porphyromonas gingivalis strains elicits a distinct effector T cell response.Cytosporone B, an inhibitor of the type III secretion system of Salmonella enterica serovar Typhimurium.Phenotypic and proteomic characterization of multiply antibiotic-resistant variants of Salmonella enterica serovar Typhimurium selected following exposure to disinfectants.A novel peptidoglycan D,L-endopeptidase induced by Salmonella inside eukaryotic cells contributes to virulence.Serratia marcescens ShlA pore-forming toxin is responsible for early induction of autophagy in host cells and is transcriptionally regulated by RcsB.Aeromonas hydrophila AH-3 type III secretion system expression and regulatory network.Unsaturated long chain free fatty acids are input signals of the Salmonella enterica PhoP/PhoQ regulatory system.A thin-layer chromatography autographic method for the detection of inhibitors of the Salmonella PhoP-PhoQ regulatory system.HilD and PhoP independently regulate the expression of grhD1, a novel gene required for Salmonella Typhimurium invasion of host cells.A protein secreted by the type III secretion system controls needle filament assemblyTyping of Salmonella Typhi strains isolated from Egypt by RAPD PCR
P2860
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P2860
PhoP-induced genes within Salmonella pathogenicity island 1.
description
2006 nî lūn-bûn
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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
PhoP-induced genes within Salmonella pathogenicity island 1.
@ast
PhoP-induced genes within Salmonella pathogenicity island 1.
@en
type
label
PhoP-induced genes within Salmonella pathogenicity island 1.
@ast
PhoP-induced genes within Salmonella pathogenicity island 1.
@en
prefLabel
PhoP-induced genes within Salmonella pathogenicity island 1.
@ast
PhoP-induced genes within Salmonella pathogenicity island 1.
@en
P2093
P2860
P356
P1476
PhoP-induced genes within Salmonella pathogenicity island 1.
@en
P2093
Andrés Aguirre
Eleonora García Véscovi
Fernando C Soncini
María Laura Cabeza
Silvana V Spinelli
P2860
P304
P356
10.1128/JB.00804-06
P407
P577
2006-10-01T00:00:00Z