Viral replication and innate host responses in primary human alveolar epithelial cells and alveolar macrophages infected with influenza H5N1 and H1N1 viruses
about
Use of ex vivo and in vitro cultures of the human respiratory tract to study the tropism and host responses of highly pathogenic avian influenza A (H5N1) and other influenza virusesHIV-1 and its gp120 inhibits the influenza A(H1N1)pdm09 life cycle in an IFITM3-dependent fashionEvaluation of the human adaptation of influenza A/H7N9 virus in PB2 protein using human and swine respiratory tract explant culturesVirus-infected alveolar epithelial cells direct neutrophil chemotaxis and inhibit their apoptosisInnate immune response of human alveolar macrophages during influenza A infectionIn vitro exposure system for study of aerosolized influenza virusClosely related influenza viruses induce contrasting respiratory tract immunopathologyInnate immune sensing and response to influenza.Differential Susceptibilities of Human Lung Primary Cells to H1N1 Influenza VirusesEarly apoptosis of porcine alveolar macrophages limits avian influenza virus replication and pro-inflammatory dysregulation.Highly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.An overview of the highly pathogenic H5N1 influenza virus.In situ molecular identification of the influenza A (H1N1) 2009 Neuraminidase in patients with severe and fatal infections during a pandemic in Mexico CityAsparagine substitution at PB2 residue 701 enhances the replication, pathogenicity, and transmission of the 2009 pandemic H1N1 influenza A virus.CTen: a web-based platform for identifying enriched cell types from heterogeneous microarray data.Multi-spectral fluorescent reporter influenza viruses (Color-flu) as powerful tools for in vivo studiesTropism of avian influenza A (H5N1) virus to mesenchymal stem cells and CD34+ hematopoietic stem cells.Construction of the influenza A virus infection-induced cell-specific inflammatory regulatory network based on mutual information and optimizationAIM2 Inflammasome Is Critical for Influenza-Induced Lung Injury and MortalitySoluble, but not transmembrane, TNF-α is required during influenza infection to limit the magnitude of immune responses and the extent of immunopathologySystems-based approach to examine the cytokine responses in primary mouse lung macrophages infected with low pathogenic avian Influenza virus circulating in South East Asia.PI3K/Akt signaling pathway modulates influenza virus induced mouse alveolar macrophage polarization to M1/M2b.Annexin V incorporated into influenza virus particles inhibits gamma interferon signaling and promotes viral replicationProinflammatory cytokine response and viral replication in mouse bone marrow derived macrophages infected with influenza H1N1 and H5N1 viruses.Immunohistochemical detection of KI polyomavirus in lung and spleen.Infection of mice with influenza A/WSN/33 (H1N1) virus alters alveolar type II cell phenotype.Host regulatory network response to infection with highly pathogenic H5N1 avian influenza virus.Endogenous Murine BST-2/Tetherin Is Not a Major Restriction Factor of Influenza A Virus Infection.Within-Host Models of High and Low Pathogenic Influenza Virus Infections: The Role of MacrophagesSusceptibility of bone marrow-derived macrophages to influenza virus infection is dependent on macrophage phenotype.The hemagglutinin protein of highly pathogenic H5N1 influenza viruses overcomes an early block in the replication cycle to promote productive replication in macrophages.Compromised respiratory function in lethal influenza infection is characterized by the depletion of type I alveolar epithelial cells beyond threshold levels.Differentiated phenotypes of primary murine alveolar epithelial cells and their susceptibility to infection by respiratory viruses.The battle between influenza and the innate immune response in the human respiratory tract.Key role of Toll-like receptor 2 in the inflammatory response and major histocompatibility complex class ii downregulation in Brucella abortus-infected alveolar macrophages.Influenza Virus Overcomes Cellular Blocks To Productively Replicate, Impacting Macrophage FunctionToll-like receptor 10 is involved in induction of innate immune responses to influenza virus infection.Cell-surface receptors on macrophages and dendritic cells for attachment and entry of influenza virus.The amazing innate immune response to influenza A virus infection.The contributions of lung macrophage and monocyte heterogeneity to influenza pathogenesis.
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P2860
Viral replication and innate host responses in primary human alveolar epithelial cells and alveolar macrophages infected with influenza H5N1 and H1N1 viruses
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@ast
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@en
type
label
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@ast
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@en
prefLabel
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@ast
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@en
P2093
P2860
P356
P1433
P1476
Viral replication and innate h ...... nfluenza H5N1 and H1N1 viruses
@en
P2093
Emily A Travanty
Jieru Wang
John M Nicholls
Michael C W Chan
Renee W Y Chan
Robert J Mason
Wendy C L Yu
P2860
P304
P356
10.1128/JVI.02200-10
P407
P50
P577
2011-05-04T00:00:00Z