The sclerostin-independent bone anabolic activity of intermittent PTH treatment is mediated by T-cell-produced Wnt10b
about
Parathyroid hormone: anabolic and catabolic actions on the skeletonT-cells and B-cells in osteoporosisT cell-expressed CD40L potentiates the bone anabolic activity of intermittent PTH treatment.Genome-Wide Mapping and Interrogation of the Nmp4 Antianabolic Bone Axis.T cells, osteoblasts, and osteocytes: interacting lineages key for the bone anabolic and catabolic activities of parathyroid hormone.Hydrogen Sulfide Is a Novel Regulator of Bone Formation Implicated in the Bone Loss Induced by Estrogen Deficiency.Macrophages: Their Emerging Roles in Bone.Sex steroid deficiency-associated bone loss is microbiota dependent and prevented by probioticsOsteoimmunology and Beyond.Physiological and pathophysiological bone turnover - role of the immune system.Hormonal and systemic regulation of sclerostin.Latest perspectives on macrophages in bone homeostasis.One Year of Abaloparatide, a Selective Activator of the PTH1 Receptor, Increased Bone Formation and Bone Mass in Osteopenic Ovariectomized Rats Without Increasing Bone Resorption.N-cadherin restrains PTH repressive effects on sclerostin/SOST by regulating LRP6-PTH1R interaction.Switching from tenofovir to abacavir in HIV-1-infected patients with low bone mineral density: changes in bone turnover markers and circulating sclerostin levels.Regulatory T cells are expanded by Teriparatide treatment in humans and mediate intermittent PTH-induced bone anabolism in mice.CTLA-4Ig (abatacept) balances bone anabolic effects of T cells and Wnt-10b with antianabolic effects of osteoblastic sclerostin.Enhanced immunoprotective effects by anti-IL-17 antibody translates to improved skeletal parameters under estrogen deficiency compared with anti-RANKL and anti-TNF-α antibodies.Treatment with intermittent PTH increases Wnt10b production by T cells in osteoporotic patients.Parathyroid Hormone Signaling in Osteocytes
P2860
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P2860
The sclerostin-independent bone anabolic activity of intermittent PTH treatment is mediated by T-cell-produced Wnt10b
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@ast
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@en
type
label
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@ast
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@en
prefLabel
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@ast
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@en
P2093
P2860
P356
P1476
The sclerostin-independent bon ...... ated by T-cell-produced Wnt10b
@en
P2093
Abdul Malik Tyagi
Jonathan Adams
Lindsey D Walker
M Neale Weitzmann
Roberto Pacifici
P2860
P356
10.1002/JBMR.2044
P577
2014-01-01T00:00:00Z