White matter injury due to experimental chronic cerebral hypoperfusion is associated with C5 deposition. - Wikidata - wikimedia - TriplyDB

White matter injury due to experimental chronic cerebral hypoperfusion is associated with C5 deposition.

White matter injury due to experimental chronic cerebral hypoperfusion is associated with C5 deposition.

http://www.wikidata.org/entity/Q35081055

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Hemoglobin and mean platelet volume predicts diffuse T1-MRI white matter volume decrease in sickle cell disease patients.Remote ischemic postconditioning: harnessing endogenous protection in a murine model of vascular cognitive impairment Stroke Damage Is Exacerbated by Nano-Size Particulate Matter in a Mouse Model.Dimethyl fumarate improves white matter function following severe hypoperfusion: Involvement of microglia/macrophages and inflammatory mediators.Experimental chronic cerebral hypoperfusion results in decreased pericyte coverage and increased blood-brain barrier permeability in the corpus callosum.Chronic cerebral hypoperfusion induced by bilateral carotid artery stenosis causes selective recognition impairment in adult mice.Estradiol Protects White Matter of Male C57BL6J Mice against Experimental Chronic Cerebral Hypoperfusion.

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White matter injury due to experimental chronic cerebral hypoperfusion is associated with C5 deposition.

http://www.wikidata.org/entity/Q35081055

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2013 nî lūn-bûn

@nan

2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

White matter injury due to exp ...... associated with C5 deposition.

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White matter injury due to exp ...... associated with C5 deposition.

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type

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White matter injury due to exp ...... associated with C5 deposition.

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White matter injury due to exp ...... associated with C5 deposition.

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prefLabel

White matter injury due to exp ...... associated with C5 deposition.

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White matter injury due to exp ...... associated with C5 deposition.

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White matter injury due to exp ...... associated with C5 deposition.

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David Shaked

http://www.w3.org/2001/XMLSchema#string

Jonathan Russin

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Leonid Groysman

http://www.w3.org/2001/XMLSchema#string

Qinghai Liu

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Steven Cen

http://www.w3.org/2001/XMLSchema#string

Thomas C Scotton

http://www.w3.org/2001/XMLSchema#string

William J Mack

http://www.w3.org/2001/XMLSchema#string

P2860

Complement inhibition promotes endogenous neurogenesis and sustained anti-inflammatory neuroprotection following reperfused stroke

Alternative pathway activation of complement by Shiga toxin promotes exuberant C3a formation that triggers microvascular thrombosis

Pattern of cerebral hypoperfusion in Alzheimer disease and mild cognitive impairment measured with arterial spin-labeling MR imaging: initial experience.

White matter lesions and glial activation in a novel mouse model of chronic cerebral hypoperfusion.

Structure of the murine fifth complement component (C5) gene. A large, highly interrupted gene with a variant donor splice site and organizational homology with the third and fourth complement component genes.

Intensity of chronic cerebral hypoperfusion determines white/gray matter injury and cognitive/motor dysfunction in mice.

Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of Alzheimer's disease

Deficiency of the murine fifth complement component (C5). A 2-base pair gene deletion in a 5'-exon.

No-reflow phenomenon.

Alzheimer's disease is a vasocognopathy: a new term to describe its nature.

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