Suppression of interferon lambda signaling by SOCS-1 results in their excessive production during influenza virus infection
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Avian Tembusu virus infection effectively triggers host innate immune response through MDA5 and TLR3-dependent signaling pathwayslncRNAs regulate the innate immune response to viral infectionInduction of innate immunity and its perturbation by influenza virusesIFN-λ: A New Inducer of Local Immunity against Cancer and InfectionsPeg-Interferon Lambda Treatment Induces Robust Innate and Adaptive Immunity in Chronic Hepatitis B Patients.Influenza A virus-induced degradation of eukaryotic translation initiation factor 4B contributes to viral replication by suppressing IFITM3 protein expression.A critical role of CDKN3 in Bcr-Abl-mediated tumorigenesisSuppressor of cytokine signaling 4 (SOCS4) protects against severe cytokine storm and enhances viral clearance during influenza infection.Expression of suppressor of cytokine signaling 1 (SOCS1) impairs viral clearance and exacerbates lung injury during influenza infection.Robust expression of vault RNAs induced by influenza A virus plays a critical role in suppression of PKR-mediated innate immunity.Targeted disruption of influenza A virus hemagglutinin in genetically modified mice reduces viral replication and improves disease outcome.Robust Lys63-Linked Ubiquitination of RIG-I Promotes Cytokine Eruption in Early Influenza B Virus Infection.Interferon Lambda: Modulating Immunity in Infectious Diseases.The regulation of autophagy by influenza A virus.Innate Immune Responses of Bat and Human Cells to Filoviruses: Commonalities and Distinctions.SOCS1 is an inducible negative regulator of IFN lambda induced gene expression in vivo.Evolution of Influenza A Virus by Mutation and Re-Assortment.SOCS4 is dispensable for an efficient recall response to influenza despite being required for primary immunity.Correction: Suppression of Interferon Lambda Signaling by SOCS-1 Results in Their Excessive Production during Influenza Virus Infection.Interferon Lambda Genetics and Biology in Regulation of Viral Control.Infection with Classical Swine Fever Virus Induces Expression of Type III Interferons and Activates Innate Immune Signaling.Differential responses of innate immunity triggered by different subtypes of influenza a viruses in human and avian hosts.Adenovirus infection of human enteroids reveals interferon sensitivity and preferential infection of goblet cells.Host Immune Response to Influenza A Virus Infection.Differential responses by human respiratory epithelial cell lines to respiratory syncytial virus reflect distinct patterns of infection control.
P2860
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P2860
Suppression of interferon lambda signaling by SOCS-1 results in their excessive production during influenza virus infection
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Suppression of interferon lamb ...... ring influenza virus infection
@ast
Suppression of interferon lamb ...... ring influenza virus infection
@en
type
label
Suppression of interferon lamb ...... ring influenza virus infection
@ast
Suppression of interferon lamb ...... ring influenza virus infection
@en
prefLabel
Suppression of interferon lamb ...... ring influenza virus infection
@ast
Suppression of interferon lamb ...... ring influenza virus infection
@en
P2093
P2860
P1433
P1476
Suppression of interferon lamb ...... ring influenza virus infection
@en
P2093
George F Gao
Ji-Long Chen
Lianfeng Zhang
Qinghuang Chen
Xiaojuan Chi
Yuhai Chen
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003845
P577
2014-01-02T00:00:00Z