Mechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression. - Wikidata - wikimedia - TriplyDB

Mechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression.

Mechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression.

http://www.wikidata.org/entity/Q35082366

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The dual regulatory role of miR-204 in cancer A dual yet opposite growth-regulating function of miR-204 and its target XRN1 in prostate adenocarcinoma cells and neuroendocrine-like prostate cancer cells E74-like factor inhibition induces reacquisition of hormone sensitiveness decreasing period circadian protein homolog 1 expression in prostate cancer cells.The NLR-related protein NWD1 is associated with prostate cancer and modulates androgen receptor signaling.SPDEF inhibits prostate carcinogenesis by disrupting a positive feedback loop in regulation of the Foxm1 oncogene.MiR-204 inhibits human NSCLC metastasis through suppression of NUAK1.Oncogenic ETS proteins mimic activated RAS/MAPK signaling in prostate cells.High SPDEF may identify patients who will have a prolonged response to androgen deprivation therapy.Relative mRNA expression of prostate-derived E-twenty-six factor and E-twenty-six variant 4 transcription factors, and of uridine phosphorylase-1 and thymidine phosphorylase enzymes, in benign and malignant prostatic tissue.The transcription factor SPDEF suppresses prostate tumor metastasis A Comprehensive NGS Data Analysis of Differentially Regulated miRNAs, piRNAs, lncRNAs and sn/snoRNAs in Triple Negative Breast Cancer.The transcription factor sterile alpha motif (SAM) pointed domain-containing ETS transcription factor (SPDEF) is required for E-cadherin expression in prostate cancer cells.Genome-wide identification of target genes for miR-204 and miR-211 identifies their proliferation stimulatory role in breast cancer cells GADD45α and γ interaction with CDK11p58 regulates SPDEF protein stability and SPDEF-mediated effects on cancer cell migration SPDEF functions as a colorectal tumor suppressor by inhibiting β-catenin activity.Role of microRNAs in lung development and pulmonary diseases miR-486-3p, miR-139-5p, and miR-21 as Biomarkers for the Detection of Oral Tongue Squamous Cell Carcinoma.PDEF in prostate cancer.Micro-RNA-204 Participates in TMPRSS2/ERG Regulation and Androgen Receptor Reprogramming in Prostate Cancer.Signatures of prostate-derived Ets factor (PDEF) in cancer.miR-204 is dysregulated in metastatic prostate cancer in vitro.microRNA-204 modulates chemosensitivity and apoptosis of prostate cancer cells by targeting zinc-finger E-box-binding homeobox 1 (ZEB1).Prostate-derived ETS factor improves prognosis and represses proliferation and invasion in hepatocellular carcinoma.A tight junction between E-Cadherin and the prostate tumor suppressor SPDEF RAS/ERK pathway transcriptional regulation through ETS/AP-1 binding sites.Nephroblastomas show low expression of microR-204 and high expression of its target, the oncogenic transcription factor MEIS1.miR-204 Shifts the Epithelial to Mesenchymal Transition in Concert with the Transcription Factors RUNX2, ETS1, and cMYB in Prostate Cancer Cell Line Model

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Mechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression.

http://www.wikidata.org/entity/Q35082366

description

2011 nî lūn-bûn

@nan

2011 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Mechanisms and functional cons ...... g prostate cancer progression.

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Mechanisms and functional cons ...... g prostate cancer progression.

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type

label

Mechanisms and functional cons ...... g prostate cancer progression.

@ast

Mechanisms and functional cons ...... g prostate cancer progression.

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prefLabel

Mechanisms and functional cons ...... g prostate cancer progression.

@ast

Mechanisms and functional cons ...... g prostate cancer progression.

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Mechanisms and functional cons ...... ng prostate cancer progression

@en

P2093

Amanda C LaRue

http://www.w3.org/2001/XMLSchema#string

David P Turner

http://www.w3.org/2001/XMLSchema#string

Dennis K Watson

http://www.w3.org/2001/XMLSchema#string

Mohamed M Desouki

http://www.w3.org/2001/XMLSchema#string

Omar Moussa

http://www.w3.org/2001/XMLSchema#string

Patricia M Watson

http://www.w3.org/2001/XMLSchema#string

Victor I Semenchenko

http://www.w3.org/2001/XMLSchema#string

P2860

PDEF, a novel prostate epithelium-specific ets transcription factor, interacts with the androgen receptor and activates prostate-specific antigen gene expression

Aberrant expression of the transcription factors snail and slug alters the response to genotoxic stress

Epithelial-mesenchymal transitions in development and disease

c-Myc-regulated microRNAs modulate E2F1 expression

MicroRNA expression profiles classify human cancers

Androgen regulation of prostasin gene expression is mediated by sterol-regulatory element-binding proteins and SLUG

Global gene expression analysis identifies PDEF transcriptional networks regulating cell migration during cancer progression

Network modeling identifies molecular functions targeted by miR-204 to suppress head and neck tumor metastasis.

Prostate-derived Ets transcription factor (PDEF) downregulates survivin expression and inhibits breast cancer cell growth in vitro and xenograft tumor formation in vivo.

Prostate-derived Ets transcription factor as a favorable prognostic marker in ovarian cancer patients.

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1723-1735

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10.1002/PROS.21389

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16

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71

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Victoria J. Findlay

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2011-03-28T00:00:00Z

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50893057

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3128180

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