Caveolae participate in tumor necrosis factor receptor 1 signaling and internalization in a human endothelial cell line.
about
CARP-2 is an endosome-associated ubiquitin ligase for RIP and regulates TNF-induced NF-kappaB activationCytokine-mediated blood brain barrier disruption as a conduit for cancer/chemotherapy-associated neurotoxicity and cognitive dysfunction.Aldosterone's rapid, nongenomic effects are mediated by striatin: a modulator of aldosterone's effect on estrogen action.Endothelial caveolin-1 regulates pathologic angiogenesis in a mouse model of colitis.TNFalpha trafficking in cerebral vascular endothelial cells.Peripheral inflammation induces tumor necrosis factor dependent AMPA receptor trafficking and Akt phosphorylation in spinal cord in addition to pain behaviorQuercetin blocks caveolae-dependent pro-inflammatory responses induced by co-planar PCBs.Polychlorinated biphenyl-induced VCAM-1 expression is attenuated in aortic endothelial cells isolated from caveolin-1 deficient mice.Targeting of tumor necrosis factor receptor 1 to low density plasma membrane domains in human endothelial cells.Caveolin-1 regulates Rac1 activation and rat pulmonary microvascular endothelial hyperpermeability induced by TNF-α.Activation of TNFR1 ectodomain shedding by mitochondrial Ca2+ determines the severity of inflammation in mouse lung microvessels.Anti-inflammatory effect of targeted delivery of SOD to endothelium: mechanism, synergism with NO donors and protective effects in vitro and in vivo.Caveolin-1 and force regulation in porcine airway smooth muscle.Translocation of endothelial nitric-oxide synthase involves a ternary complex with caveolin-1 and NOSTRIN.Plasma membrane microdomains regulate TACE-dependent TNFR1 shedding in human endothelial cells.Tumour necrosis factor receptor trafficking dysfunction opens the TRAPS door to pro-inflammatory cytokine secretion.Generation of NO by bystander human CD8 T cells augments allogeneic responses by inhibiting cytokine deprivation-induced cell deathCaveolae-mediated internalization of occludin and claudin-5 during CCL2-induced tight junction remodeling in brain endothelial cells.Active uptake of dendritic cell-derived exovesicles by epithelial cells induces the release of inflammatory mediators through a TNF-alpha-mediated pathway.IFN-alpha induces transcription of hypoxia-inducible factor-1alpha to inhibit proliferation of human endothelial cells.LRRC8A channels support TNFα-induced superoxide production by Nox1 which is required for receptor endocytosis.Cytokine signaling modulates blood-brain barrier function.Sepsis-Associated Encephalopathy: The Blood-Brain Barrier and the Sphingolipid Rheostat.Knock down of caveolin-1 affects morphological and functional hallmarks of human endothelial cells.Influence of chimeric human-bovine fibers on adenoviral uptake by liver cells and the antiviral immune response.Dynamics of RASSF1A/MOAP-1 association with death receptors.Caveolin mediates rapid glucocorticoid effects and couples glucocorticoid action to the antiproliferative program.Apoptotic phenotype alters the capacity of tumor necrosis factor-related apoptosis-inducing ligand to induce human vascular endothelial activation.Dispensability and dynamics of caveolin-1 during liver regeneration and in isolated hepatic cells.Mechanism for removal of tumor necrosis factor receptor 1 from the cell surface by the adenovirus RIDalpha/beta complex.Cavin-2 Functions as a Suppressive Regulator in TNF-induced Mesenchymal Stromal Cell Inflammation and Angiogenic Phenotypes.Spatial segregation of transport and signalling functions between human endothelial caveolae and lipid raft proteomes.The tumour suppressor Ras-association domain family protein 1A (RASSF1A) regulates TNF-α signalling in cardiomyocytes.Endothelial caveolae and caveolin-1 as key regulators of atherosclerosis.The role of fatty acids and caveolin-1 in tumor necrosis factor alpha-induced endothelial cell activation.Both internalization and AIP1 association are required for tumor necrosis factor receptor 2-mediated JNK signaling.Caveolae: a regulatory platform for nutritional modulation of inflammatory diseasesSpatial compartmentalization of tumor necrosis factor (TNF) receptor 1-dependent signaling pathways in human airway smooth muscle cells. Lipid rafts are essential for TNF-alpha-mediated activation of RhoA but dispensable for the activation of the NFIL-6/sIL-6R enhances cathepsin B and L production via caveolin-1-mediated JNK-AP-1 pathway in human gingival fibroblasts.The MAP kinase pathway mediates transcytosis induced by TNF-alpha in an in vitro blood-brain barrier model.
P2860
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P2860
Caveolae participate in tumor necrosis factor receptor 1 signaling and internalization in a human endothelial cell line.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Caveolae participate in tumor ...... a human endothelial cell line.
@ast
Caveolae participate in tumor ...... a human endothelial cell line.
@en
type
label
Caveolae participate in tumor ...... a human endothelial cell line.
@ast
Caveolae participate in tumor ...... a human endothelial cell line.
@en
prefLabel
Caveolae participate in tumor ...... a human endothelial cell line.
@ast
Caveolae participate in tumor ...... a human endothelial cell line.
@en
P2093
P2860
P1476
Caveolae participate in tumor ...... a human endothelial cell line.
@en
P2093
Alessio D'Alessio
John R Bradley
Jordan S Pober
Rafia S Al-Lamki
P2860
P304
P356
10.1016/S0002-9440(10)62346-2
P407
P577
2005-04-01T00:00:00Z