Lack of tissue inhibitor of metalloproteinases-3 results in an enhanced inflammatory response in antigen-induced arthritis.
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Interleukin-10 regulates TNF-alpha-converting enzyme (TACE/ADAM-17) involving a TIMP-3 dependent and independent mechanismProgress in matrix metalloproteinase research.Tissue inhibitor of metalloproteinase-3 (TIMP-3) regulates hematopoiesis and bone formation in vivo.ADAM-17: the enzyme that does it all.The tissue inhibitors of metalloproteinases (TIMPs): an ancient family with structural and functional diversityTIMP3 regulates mammary epithelial apoptosis with immune cell recruitment through differential TNF dependence.Differential regulation of matrix-metalloproteinases and their tissue inhibitors in patients with aneurysmal subarachnoid hemorrhage.IGFBP-3 inhibits TNF-α production and TNFR-2 signaling to protect against retinal endothelial cell apoptosis.Cigarette smoke components induce matrix metalloproteinase-1 in aortic endothelial cells through inhibition of mTOR signaling.Proteases involved in cartilage matrix degradation in osteoarthritis.(Make) stick and cut loose--disintegrin metalloproteases in development and disease.Intravitreal injection of TIMP3 or the EGFR inhibitor erlotinib offers protection from oxygen-induced retinopathy in mice.Inflammation and breast cancer: metalloproteinases as common effectors of inflammation and extracellular matrix breakdown in breast cancerThe role of ADAMTSs in arthritisCurrent and emerging therapeutic strategies for preventing inflammation and aggrecanase-mediated cartilage destruction in arthritis.Growth Factors in the Pathogenesis of Retinal Neurodegeneration in Diabetes MellitusPathological neovascularization is reduced by inactivation of ADAM17 in endothelial cells but not in pericytes.Involvement of a disintegrin and metalloproteinase 10 and 17 in shedding of tumor necrosis factor-alpha.ADAM17 is regulated by a rapid and reversible mechanism that controls access to its catalytic site.Macrocyclic θ-defensins suppress tumor necrosis factor-α (TNF-α) shedding by inhibition of TNF-α converting enzyme.Exercise training and return to a well-balanced diet activate the neuregulin 1/ErbB pathway in skeletal muscle of obese rats.Timp3 loss accelerates tumour invasion and increases prostate inflammation in a mouse model of prostate cancer.The EGFR-ADAM17 Axis in Chronic Obstructive Pulmonary Disease and Cystic Fibrosis Lung Pathology.The genetics of retinopathy of prematurity: a model for neovascular retinal disease
P2860
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P2860
Lack of tissue inhibitor of metalloproteinases-3 results in an enhanced inflammatory response in antigen-induced arthritis.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@ast
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@en
type
label
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@ast
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@en
prefLabel
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@ast
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@en
P2093
P2860
P1476
Lack of tissue inhibitor of me ...... in antigen-induced arthritis.
@en
P2093
David Smookler
John S Mort
Mandana Mahmoodi
Rama Khokha
Solmaz Sahebjam
P2860
P304
P356
10.1016/S0002-9440(10)62483-2
P407
P577
2005-06-01T00:00:00Z